Brailowsky S, Knight R T
Brain Res. 1984 Nov 26;322(2):310-5. doi: 10.1016/0006-8993(84)90123-9.
In anesthetized preparations, GABA and taurine produced rapid, reversible inhibition of the negative component (N20) of the primary somatosensory evoked potential (SEP) without effect on the earlier positivity (P11). This effect was also produced by low doses of 4-aminopyridine. Neither bicuculline or picrotoxin antagonized these drug effects. A predominance of type B GABA receptors in the superficial layers of the somatosensory cortex is proposed.
在麻醉制剂中，γ-氨基丁酸（GABA）和牛磺酸可快速、可逆地抑制初级体感诱发电位（SEP）的负向成分（N20），而对较早出现的正向成分（P11）无影响。低剂量的4-氨基吡啶也会产生这种效应。荷包牡丹碱或印防己毒素均不能拮抗这些药物的作用。有人提出在体感皮层表层B型GABA受体占主导地位。
