胃泌素刺激两栖类动物胃黏膜中的氢离子分泌：河豚毒素的作用。
Gastrin stimulated H+ secretion in amphibian gastric mucosa: effect of tetrodotoxin.
作者信息
Spenney J G
出版信息
Experientia. 1978 May 15;34(5):625-6. doi: 10.1007/BF01936999.
PMID:658248
Abstract
The role of neural mechanisms in gastrin stimulated H+ secretion was studied using amphibian gastric fundic mucosa. Spontaneously secreting mucosae were converted to resting state (zero H+ secretory rate) using Burimamide. Following removal of burimamide, 3 X 10(-6) M tetrodotoxin did not block gastrin stimulation of H+ secretion indicating that neural mechanisms are not required.
摘要
利用两栖类胃底黏膜研究了神经机制在胃泌素刺激氢离子分泌中的作用。使用甲氰咪胍将自发分泌的黏膜转变为静息状态（氢离子分泌率为零）。去除甲氰咪胍后，3×10⁻⁶ M的河豚毒素并未阻断胃泌素对氢离子分泌的刺激，这表明不需要神经机制。
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