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Degradation of newly synthesized collagen.

作者信息

Bienkowski R S, Cowan M J, McDonald J A, Crystal R G

出版信息

J Biol Chem. 1978 Jun 25;253(12):4356-63.

PMID:659420
Abstract
摘要

相似文献

1
Degradation of newly synthesized collagen.新合成胶原蛋白的降解
J Biol Chem. 1978 Jun 25;253(12):4356-63.
2
Ascorbate-dependent differences in the hydroxylation of proline and lysine in collagen synthesized by 3T6 fibroblasts in culture.培养的3T6成纤维细胞合成的胶原蛋白中脯氨酸和赖氨酸羟基化过程中依赖抗坏血酸的差异。
Biochim Biophys Acta. 1972 Oct 31;278(3):610-6. doi: 10.1016/0005-2795(72)90025-6.
3
Further studies on the effect of the collagen triple-helix formation on the hydroxylation of lysine and the glycosylations of hydroxylysine in chick-embryo tendon and cartilage cells.关于胶原蛋白三螺旋形成对鸡胚肌腱和软骨细胞中赖氨酸羟基化及羟赖氨酸糖基化影响的进一步研究。
Biochem J. 1977 Sep 15;166(3):357-62. doi: 10.1042/bj1660357.
4
Effect of amino acids on collagen biosynthesis.氨基酸对胶原蛋白生物合成的影响。
In Vitro. 1973 Mar-Apr;8(5):342-6. doi: 10.1007/BF02619058.
5
The synthesis of connective tissue protein in smooth muscle cells.平滑肌细胞中结缔组织蛋白的合成。
Biochim Biophys Acta. 1976 Jan 5;418(1):93-103. doi: 10.1016/0005-2787(76)90330-0.
6
[Some enzymatic stages of collagen biosynthesis].[胶原蛋白生物合成的一些酶促阶段]
Reumatologia. 1972;10(2):171-80.
7
Incorporation of cis-hydroxyproline into protocollagen and collagen. Collagen containing cis-hydroxyproline in place of proline and trans-hydroxyproline is not extruded at a normal rate.顺式羟脯氨酸掺入原胶原和胶原中。含有顺式羟脯氨酸而非脯氨酸和反式羟脯氨酸的胶原不会以正常速率挤出。
J Biol Chem. 1971 Mar 25;246(6):1549-55.
8
Inhibitory effect of L-dopa on collagen biosynthesis.
Acta Med Scand. 1974 Jan-Feb;195(1-2):37-8. doi: 10.1111/j.0954-6820.1974.tb08093.x.
9
Biosynthesis of collagen and elastin by chick embryo skin during maturation.鸡胚皮肤成熟过程中胶原蛋白和弹性蛋白的生物合成。
Arch Dermatol Forsch. 1974;249(2):99-104. doi: 10.1007/BF00595571.
10
The in vitro effect of insulin on collagen synthesis in embryonic chick tibia.
Proc Soc Exp Biol Med. 1973 Apr;142(4):1152-4. doi: 10.3181/00379727-142-37196.

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New Mechanisms for Regulation of Intracellular Collagen Degradation in Fibrotic Lesions of Periodontal Tissues.牙周组织纤维化病变中细胞内胶原降解调控的新机制
FASEB J. 2025 Oct 31;39(20):e71171. doi: 10.1096/fj.202502454R.
2
You Say You Want a Resolution (of Fibrosis).你说你想要(纤维化的)解决办法。
Am J Respir Cell Mol Biol. 2020 Oct;63(4):424-435. doi: 10.1165/rcmb.2020-0182TR.
3
Mechanisms for the Resolution of Organ Fibrosis.器官纤维化的解决机制。
Physiology (Bethesda). 2019 Jan 1;34(1):43-55. doi: 10.1152/physiol.00033.2018.
4
Linkage of bacterial protein synthesis and presentation of MHC class I-restricted Listeria monocytogenes-derived antigenic peptides.细菌蛋白质合成与 MHC I 类限制性李斯特菌衍生抗原肽呈递的关联。
PLoS One. 2012;7(3):e33335. doi: 10.1371/journal.pone.0033335. Epub 2012 Mar 12.
5
A weak link in metabolism: the metabolic capacity for glycine biosynthesis does not satisfy the need for collagen synthesis.代谢中的薄弱环节:甘氨酸生物合成的代谢能力无法满足胶原蛋白合成的需求。
J Biosci. 2009 Dec;34(6):853-72. doi: 10.1007/s12038-009-0100-9.
6
The 21-day postnatal rat ventricular cardiac muscle cell in culture as an experimental model to study adult cardiomyocyte gene expression.将培养的出生后21天的大鼠心室心肌细胞作为研究成年心肌细胞基因表达的实验模型。
Mol Cell Biochem. 2002 Jan;229(1-2):51-62. doi: 10.1023/a:1017999216277.
7
Transforming growth factor-beta initiates wound repair in rat liver through induction of the EIIIA-fibronectin splice isoform.转化生长因子-β通过诱导EIIIA-纤连蛋白剪接异构体启动大鼠肝脏的伤口修复。
Am J Pathol. 2000 Jan;156(1):115-24. doi: 10.1016/s0002-9440(10)64711-6.
8
In vivo inhibition of rat stellate cell activation by soluble transforming growth factor beta type II receptor: a potential new therapy for hepatic fibrosis.可溶性转化生长因子βⅡ型受体对大鼠星状细胞激活的体内抑制作用:肝纤维化的一种潜在新疗法。
Proc Natl Acad Sci U S A. 1999 Oct 26;96(22):12719-24. doi: 10.1073/pnas.96.22.12719.
9
Expression of variant fibronectins in wound healing: cellular source and biological activity of the EIIIA segment in rat hepatic fibrogenesis.伤口愈合中变异纤连蛋白的表达:大鼠肝纤维化形成过程中EIIIA片段的细胞来源及生物学活性
J Cell Biol. 1994 Dec;127(6 Pt 2):2037-48. doi: 10.1083/jcb.127.6.2037.
10
Cell-specific expression of transforming growth factor-beta in rat liver. Evidence for autocrine regulation of hepatocyte proliferation.转化生长因子-β在大鼠肝脏中的细胞特异性表达。肝细胞增殖自分泌调节的证据。
J Clin Invest. 1995 Jul;96(1):447-55. doi: 10.1172/JCI118055.