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Induced cell differentiation in cancer therapy.

作者信息

Bloch A

出版信息

Cancer Treat Rep. 1984 Jan;68(1):199-205.

PMID:6692427
Abstract
摘要

相似文献

1
Induced cell differentiation in cancer therapy.癌症治疗中的诱导细胞分化
Cancer Treat Rep. 1984 Jan;68(1):199-205.
2
Modulation of cellular differentiation and cell fate for cancer therapy.用于癌症治疗的细胞分化和细胞命运调控
Curr Opin Investig Drugs. 2004 Dec;5(12):1268-73.
3
[Development of differentiation therapy].[分化疗法的发展]
Gan To Kagaku Ryoho. 1993 Jun;20(8):1006-8.
4
Differentiation: a suitable strategy for cancer chemotherapy?分化:癌症化疗的合适策略?
Anticancer Drug Des. 1993 Aug;8(4):299-322.
5
Rapid assay for evaluating the chemosensitivity of human tumors in soft agar culture.在软琼脂培养中评估人类肿瘤化学敏感性的快速检测方法。
Cancer Res. 1982 Jun;42(6):2159-64.
6
Some developments in cancer chemotherapy.癌症化疗的一些进展。
Nurs Mirror. 1978 Mar 23;146(12):18-20.
7
New targets for cancer chemotherapy.癌症化疗的新靶点。
Cancer Chemother Biol Response Modif. 2002;20:239-72.
8
[Novel targets for cancer chemotherapy].[癌症化疗的新靶点]
Gan To Kagaku Ryoho. 1996 Jan;23(2):191-201.
9
IP6-induced growth inhibition and differentiation of HT-29 human colon cancer cells: involvement of intracellular inositol phosphates.肌醇六磷酸诱导HT-29人结肠癌细胞生长抑制和分化:细胞内肌醇磷酸的参与
Anticancer Res. 1995 Nov-Dec;15(6B):2479-87.
10
Synchronization of malignant cell proliferation, host rhythms and timing of cytostatic therapy.
Rev Roum Med Intern. 1975;13(1):3-11.

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Comparative proteomics and molecular mechanical analysis in CDA-II induced therapy of LCI-D20 hepatocellular carcinoma model.CDA-II诱导治疗LCI-D20肝癌模型中的比较蛋白质组学和分子力学分析
J Cancer Res Clin Oncol. 2009 Apr;135(4):591-602. doi: 10.1007/s00432-008-0493-0. Epub 2008 Oct 14.
2
Molecular Events as Targets of Anticancer Drug Therapy.作为抗癌药物治疗靶点的分子事件
Pathol Oncol Res. 1997;3(2):147-158. doi: 10.1007/BF02907812.
3
Hierarchical and cybernetic nature of biologic systems and their relevance to homeostatic adaptation to low-level exposures to oxidative stress-inducing agents.
生物系统的层次结构和控制论本质及其与对低水平氧化应激诱导剂暴露的稳态适应的相关性。
Environ Health Perspect. 1998 Feb;106 Suppl 1(Suppl 1):331-9. doi: 10.1289/ehp.98106s1331.
4
Genes related to growth and invasiveness are repressed by sodium butyrate in ovarian carcinoma cells.与生长和侵袭相关的基因在卵巢癌细胞中被丁酸钠抑制。
Br J Cancer. 1996 Feb;73(4):433-8. doi: 10.1038/bjc.1996.78.
5
Selective growth arrest and phenotypic reversion of prostate cancer cells in vitro by nontoxic pharmacological concentrations of phenylacetate.苯乙酸无毒药理浓度在体外对前列腺癌细胞的选择性生长抑制及表型逆转
J Clin Invest. 1993 May;91(5):2288-95. doi: 10.1172/JCI116457.
6
Characterization of insulin-like growth factor I binding sites in human bladder cancer cell lines.
Urol Res. 1993 Jan;21(1):27-32. doi: 10.1007/BF00295188.
7
Dimethyl sulfoxide-induced differentiation does not alter tumorigenicity of neuroblastoma cells.二甲基亚砜诱导的分化不会改变神经母细胞瘤细胞的致瘤性。
J Neurooncol. 1985;3(1):43-51. doi: 10.1007/BF00165171.
8
Salivary concentrations of hexamethylene bisacetamide (HMBA) in patients receiving 5-day continuous infusions.接受为期5天持续输注的患者唾液中六甲撑双乙酰胺(HMBA)的浓度。
Cancer Chemother Pharmacol. 1988;22(1):65-8. doi: 10.1007/BF00254184.
9
The natural history of a family of transplantable melanomas in hamsters.仓鼠可移植黑色素瘤家族的自然病史。
Cancer Metastasis Rev. 1988 Jun;7(2):95-118. doi: 10.1007/BF00046481.
10
Induction of erythroid differentiation by the anthracycline antitumor antibiotic pyrromycin.蒽环类抗肿瘤抗生素吡咯霉素诱导红系分化
Cell Biol Toxicol. 1988 Mar;4(1):123-33. doi: 10.1007/BF00141291.