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犬心肌再灌注损伤。超氧化物歧化酶和过氧化氢酶联合给药对其的减轻作用。

Canine myocardial reperfusion injury. Its reduction by the combined administration of superoxide dismutase and catalase.

作者信息

Jolly S R, Kane W J, Bailie M B, Abrams G D, Lucchesi B R

出版信息

Circ Res. 1984 Mar;54(3):277-85. doi: 10.1161/01.res.54.3.277.

DOI:10.1161/01.res.54.3.277
PMID:6697450
Abstract

Therapy directed against the toxic effects of reactive oxygen species may reduce the final extent of ischemic injury in otherwise viable tissue irreversibly injured by the abrupt reoxygenation of reperfusion. In four groups of dogs, superoxide dismutase plus catalase (groups I-III) or saline (controls) (group IV) was infused into the left atrium. Group I received the infusion for 2 hours, beginning 15 minutes before occlusion of the left circumflex coronary artery (90 minutes) and ending 15 minutes after reperfusion. Group II received the infusion for 1 hour starting 15 minutes before reperfusion. Group III received the infusion for 1 hour beginning 40 minutes after reperfusion. Dogs were killed the next day, and infarct size was determined by dissection and weighing, and confirmed histologically. Infarct size expressed as percent of the anatomic area at risk was: group I, 19.4 +/- 5.0; group II, 21.8 +/- 3.3; group III, 47.6 +/- 10.3; group IV, 43.6 +/- 3.5 (mean +/- SEM). Analysis of variance followed by Duncan's multiple range test showed that ultimate infarct size as assessed in groups I and II differed significantly (P less than 0.05) from that observed in the control animals in group IV, whereas infarct size between groups III and IV did not differ significantly (P greater than 0.05). The percent of left ventricle at risk did not differ between the four groups. The beneficial effects of superoxide dismutase plus catalase could not be explained by hemodynamic differences. Similar protection of jeopardized myocardium in groups I and II suggest that potentially viable tissue is salvaged by scavenging free radicals during early reperfusion.(ABSTRACT TRUNCATED AT 250 WORDS)

摘要

针对活性氧毒性作用的治疗可能会减少在再灌注时突然复氧而导致的原本存活组织发生不可逆损伤的最终缺血损伤范围。在四组犬中,将超氧化物歧化酶加过氧化氢酶(第一至三组)或生理盐水(对照组)(第四组)注入左心房。第一组在左回旋支冠状动脉闭塞前15分钟(共90分钟)开始注入,持续2小时,至再灌注后15分钟结束。第二组在再灌注前15分钟开始注入,持续1小时。第三组在再灌注后40分钟开始注入,持续1小时。犬于次日处死,通过解剖和称重确定梗死面积,并进行组织学确认。以危险解剖区域的百分比表示的梗死面积为:第一组,19.4±5.0;第二组,21.8±3.3;第三组,47.6±10.3;第四组,43.6±3.5(平均值±标准误)。方差分析后进行邓肯多重极差检验表明,第一组和第二组评估的最终梗死面积与第四组对照动物观察到的梗死面积有显著差异(P<0.05),而第三组和第四组之间的梗死面积无显著差异(P>0.05)。四组之间危险左心室的百分比无差异。超氧化物歧化酶加过氧化氢酶的有益作用无法用血流动力学差异来解释。第一组和第二组对濒危心肌的类似保护表明,在早期再灌注期间通过清除自由基可挽救潜在存活的组织。(摘要截断于250字)

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