Ish-Shalom N, Better O S
Isr J Med Sci. 1984 Feb;20(2):109-12.
The effect of immersion for 4 h on arterial blood pressure and the rate or urinary sodium excretion was studied in five subjects both in fresh water and in Dead Sea water (specific gravity 1.19) at 34 C. Following 100 min of immersion, mean systolic and diastolic blood pressure decreased (P less than 0.01) in subjects immersed in fresh water, and increased (P less than 0.05) in those immersed in Dead Sea water. While immersion in fresh water led to hypotension, it was associated with an increased natriuresis. In contrast, the hypertensive response to immersion in Dead Sea water was not associated with an increased natriuresis. It is concluded that under the unique conditions of this experiment, urinary excretion of sodium becomes independent of systemic arterial blood pressure, and is presumably governed by neurohumoral influences originating in the baroreceptors of the low pressure system.
在34摄氏度下，对5名受试者分别在淡水和死海水中（比重1.19）浸泡4小时对动脉血压和尿钠排泄率的影响进行了研究。浸泡100分钟后，浸泡在淡水中的受试者平均收缩压和舒张压下降（P<0.01），而浸泡在死海水中的受试者则升高（P<0.05）。虽然浸泡在淡水中会导致低血压，但它与尿钠排泄增加有关。相比之下，浸泡在死海水中的高血压反应与尿钠排泄增加无关。得出的结论是，在本实验的独特条件下，尿钠排泄变得独立于体循环动脉血压，并且可能受起源于低压系统压力感受器的神经体液影响所支配。
