Schor C
Am J Optom Physiol Opt. 1978 Mar;55(3):183-6. doi: 10.1097/00006324-197803000-00007.
A motor theory is proposed to explain monocular eccentric fixation of amblyopic eyes. Clinical observations that suggest the involvement of a motor anomaly related to strabismus are cited. The model proposes that potentiation (after discharge) of the agonist muscle in strabismus results in a nonregistered error of monocular fixation of the amblyopic eye. The position error goes undetected as a result of reduced sensitivity of the amblyopic eye. Evidence is cited for normal muscle potentiation in nonamblyopic eyes. A suggestion is made that amblyopia be considered both a sensory and a motor disorder. The sensory disorder is the reduced acuity at the fovea of the amblyopic eye. The motor disorder is muscle potentiation that causes the amblyopic eye to fixate with an eccentric retinal region that has lower acuity than the fovea. A distinction is made between motor anomalies that have a sensory basis, such as unsteady fixation in anisometropic amblyopia, and motor anomalies that have a motor basis, such as eccentric fixation in strabismic amblyopia.
提出了一种运动理论来解释弱视眼的单眼偏心注视。文中引用了临床观察结果，这些结果表明存在与斜视相关的运动异常。该模型提出，斜视中主动肌的增强（后放电）会导致弱视眼单眼注视出现未被记录的误差。由于弱视眼敏感性降低，位置误差未被察觉。文中引用了非弱视眼正常肌肉增强的证据。有人提出，弱视应被视为一种感觉和运动障碍。感觉障碍是弱视眼中央凹视力下降。运动障碍是肌肉增强，导致弱视眼用视网膜偏心区域注视，该区域视力低于中央凹。文中区分了具有感觉基础的运动异常，如屈光参差性弱视中的不稳定注视，以及具有运动基础的运动异常，如斜视性弱视中的偏心注视。
