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Relationship of the single-electron reduction potential of quinones to their reduction by flavoproteins.

作者信息

Powis G, Appel P L

出版信息

Biochem Pharmacol. 1980 Oct 1;29(19):2567-72. doi: 10.1016/0006-2952(80)90068-4.

DOI:10.1016/0006-2952(80)90068-4
PMID:6775639
Abstract
摘要

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Relationship of the single-electron reduction potential of quinones to their reduction by flavoproteins.醌类的单电子还原电位与其被黄素蛋白还原之间的关系。
Biochem Pharmacol. 1980 Oct 1;29(19):2567-72. doi: 10.1016/0006-2952(80)90068-4.
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Quinones and glutathione metabolism.
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[Ubiquinone content and the oxidative-reductive enzymatic system activity in the liver of vitamin E-deficient rats administered alpha-tocopherol and its chlorine derivative].[给予α-生育酚及其氯衍生物的维生素E缺乏大鼠肝脏中的泛醌含量及氧化还原酶系统活性]
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Genetic evidence for NAD(P)H:quinone oxidoreductase 1-catalyzed quinone reduction on passage through the mouse pulmonary circulation.遗传证据表明 NAD(P)H:醌氧化还原酶 1 在穿过小鼠肺循环时催化醌还原。
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Hemorrhage increases cytokine expression in lung mononuclear cells in mice: involvement of catecholamines in nuclear factor-kappaB regulation and cytokine expression.出血增加小鼠肺单核细胞中细胞因子的表达:儿茶酚胺参与核因子-κB的调节及细胞因子表达。
J Clin Invest. 1997 Apr 1;99(7):1516-24. doi: 10.1172/JCI119314.
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Enzymology of bioreductive drug activation.生物还原药物激活的酶学
Br J Cancer Suppl. 1996 Jul;27:S1-8.
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Inhibition of relaxations to nitrergic stimulation of the mouse anococcygeus by duroquinone.杜罗醌对小鼠肛门尾骨肌硝化能刺激引起的舒张的抑制作用。
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Selective inhibition of adenylate cyclase in bovine cortex by quinones: a novel cellular substrate for quinone cytotoxicity.醌类对牛皮层中腺苷酸环化酶的选择性抑制:醌类细胞毒性的一种新的细胞底物。
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Reduction of ubiquinone in membrane lipids by rat liver cytosol and its involvement in the cellular defence system against lipid peroxidation.大鼠肝细胞溶胶对膜脂中泛醌的还原作用及其在细胞抗脂质过氧化防御系统中的作用。
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Toxic drug effects associated with oxygen metabolism: redox cycling and lipid peroxidation.与氧代谢相关的毒性药物作用:氧化还原循环和脂质过氧化。
Experientia. 1981 Dec 15;37(12):1233-41. doi: 10.1007/BF01948335.
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Soft-tissue injury caused by antineoplastic drugs is inhibited by topical dimethyl sulphoxide and alpha tocopherol.局部使用二甲基亚砜和α-生育酚可抑制抗肿瘤药物引起的软组织损伤。
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