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Complement lysis of human erythrocytes. III. Differing effectiveness of human and guinea pig C9 on normal and paroxysmal nocturnal hemoglobinuria cells.

作者信息

Rosenfeld S I, Packman C H, Jenkins D E, Countryman J K, Leddy J P

出版信息

J Immunol. 1980 Nov;125(5):2063-8.

PMID:6776186
Abstract
摘要

相似文献

1
Complement lysis of human erythrocytes. III. Differing effectiveness of human and guinea pig C9 on normal and paroxysmal nocturnal hemoglobinuria cells.人红细胞的补体溶解作用。III. 人及豚鼠C9对正常和阵发性夜间血红蛋白尿细胞的不同作用效果
J Immunol. 1980 Nov;125(5):2063-8.
2
Complement lysis of human erythrocytes. Differeing susceptibility of two types of paroxysmal nocturnal hemoglobinuria cells to C5b-9.人红细胞的补体溶解。两种阵发性夜间血红蛋白尿细胞对C5b-9的不同敏感性。
J Clin Invest. 1979 Aug;64(2):428-33. doi: 10.1172/JCI109479.
3
Complement lysis of human erythrocytes. II. A unique interaction of human C8 and C9 with paroxysmal nocturnal hemoglobinuria erythrocytes.人红细胞的补体溶解作用。II. 人C8和C9与阵发性夜间血红蛋白尿症红细胞的独特相互作用。
J Immunol. 1980 Jun;124(6):2818-23.
4
Enhanced reactive lysis of paroxysmal nocturnal hemoglobinuria erythrocytes by C5b-9 does not involve increased C7 binding or cell-bound C3b.C5b-9对阵发性夜间血红蛋白尿红细胞的反应性溶解增强并不涉及C7结合增加或细胞结合的C3b增加。
J Immunol. 1985 Jan;134(1):506-11.
5
Activation of the fifth and sixth component of the complement system: similarities between C5b6 and C(56)a with respect to lytic enhancement by cell-bound C3b or A2C, and species preferences of target cell.补体系统第五和第六成分的激活：C5b6与C(56)a在细胞结合的C3b或A2C介导的溶解增强方面的相似性以及靶细胞的物种偏好
J Immunol. 1981 Sep;127(3):999-1002.
6
Lytic activity of C5-9 complexes for erythrocytes from the species other than sheep: C9 rather than C8-dependent variation in lytic activity.C5 - 9复合物对绵羊以外物种红细胞的溶解活性：溶解活性中依赖C9而非C8的变化。
J Immunol. 1977 Oct;119(4):1482-5.
7
Paroxysmal nocturnal hemoglobinuria type III. Lack of an erythrocyte membrane protein restricting the lysis by C5b-9.III型阵发性夜间血红蛋白尿。缺乏限制C5b-9介导红细胞溶解的红细胞膜蛋白。
J Clin Invest. 1987 Jul;80(1):7-12. doi: 10.1172/JCI113065.
8
Enhanced complement-mediated lysis of type III paroxysmal nocturnal hemoglobinuria erythrocytes involves increased C9 binding and polymerization.补体介导的Ⅲ型阵发性夜间血红蛋白尿红细胞溶解增强涉及C9结合和聚合增加。
Proc Natl Acad Sci U S A. 1985 Aug;82(16):5520-4. doi: 10.1073/pnas.82.16.5520.
9
Enhanced reactive lysis of paroxysmal nocturnal hemoglobinuria erythrocytes. Studies on C9 binding and incorporation into high molecular weight complexes.阵发性夜间血红蛋白尿红细胞的增强反应性溶解。关于C9结合及掺入高分子量复合物的研究。
J Exp Med. 1986 Oct 1;164(4):981-97. doi: 10.1084/jem.164.4.981.
10
Trypsin-activated complex of human factor B with cobra venom factor (CVF), cleaving C3 and C5 and generating a lytic factor for unsensitized guinea pig erythrocytes. I. Generation of the activated complex.人补体B因子与眼镜蛇毒因子（CVF）的胰蛋白酶激活复合物，可裂解C3和C5，并产生针对未致敏豚鼠红细胞的溶血因子。I. 激活复合物的产生。
Biken J. 1975 Dec;18(4):193-204.

引用本文的文献

1
Homologous species restriction in lysis of erythrocytes by terminal complement proteins.终末补体蛋白对红细胞溶解的同源物种限制
Proc Natl Acad Sci U S A. 1981 Aug;78(8):5118-21. doi: 10.1073/pnas.78.8.5118.
2
Inhibition of the lytic action of cell-bound terminal complement components by human high density lipoproteins and apoproteins.人高密度脂蛋白及载脂蛋白对细胞结合的末端补体成分溶解作用的抑制
J Clin Invest. 1983 Apr;71(4):795-808. doi: 10.1172/jci110833.
3
Enhanced complement-mediated lysis of type III paroxysmal nocturnal hemoglobinuria erythrocytes involves increased C9 binding and polymerization.
补体介导的Ⅲ型阵发性夜间血红蛋白尿红细胞溶解增强涉及C9结合和聚合增加。
Proc Natl Acad Sci U S A. 1985 Aug;82(16):5520-4. doi: 10.1073/pnas.82.16.5520.
4
Inhibition of antibody-dependent lymphocyte cytotoxicity by homologous restriction factor incorporated into target cell membranes.掺入靶细胞膜中的同源限制因子对抗体依赖性淋巴细胞细胞毒性的抑制作用。
J Exp Med. 1987 Oct 1;166(4):947-55. doi: 10.1084/jem.166.4.947.
5
Paroxysmal nocturnal hemoglobinuria type III. Lack of an erythrocyte membrane protein restricting the lysis by C5b-9.III型阵发性夜间血红蛋白尿。缺乏限制C5b-9介导红细胞溶解的红细胞膜蛋白。
J Clin Invest. 1987 Jul;80(1):7-12. doi: 10.1172/JCI113065.
6
Deficiency of the homologous restriction factor in paroxysmal nocturnal hemoglobinuria.阵发性夜间血红蛋白尿中同源限制因子的缺乏
J Exp Med. 1987 Feb 1;165(2):572-7. doi: 10.1084/jem.165.2.572.
7
Isolation of a human erythrocyte membrane protein capable of inhibiting expression of homologous complement transmembrane channels.一种能够抑制同源补体跨膜通道表达的人红细胞膜蛋白的分离。
Proc Natl Acad Sci U S A. 1986 Sep;83(18):6975-9. doi: 10.1073/pnas.83.18.6975.