Szibor R, Till U, Lösche W, Steinbicker V
Acta Biol Med Ger. 1981;40(9):1187-90.
Treatment of red cells from Duchenne muscular dystrophy (DMD) patients with A23187 induced identical responses with those in controls in terms of Ca++ flux, shrinkage, ATP depletion, hypotonic lysis protection, and glycolysis stimulation. The level of RC-free calcium seemed not to be altered in DMD. Valinomycine-induced shrinkage was enhanced significantly in DMD red blood cells.
