Dietz V, Berger W
Exp Neurol. 1983 Mar;79(3):680-7. doi: 10.1016/0014-4886(83)90032-8.
The activation of leg muscles was analyzed in respect to ankle joint movement and the changes in tension produced by the triceps surae muscle during slow gait in spastic adults and children with cerebral palsy. In normal subjects the increase in tension of the triceps surae in the stance phase of gait is mainly due to an increase in gastrocnemius and soleus EMG. In spastic patients the abnormally high tension development in triceps surae is due more to passive muscle stretch, for the reciprocally organized leg muscle EMG is reduced. It is concluded that the leg extensor muscles in spastic patients exhibit a pseudostretch-reflex behavior due to their mechanical properties, and that this is mainly responsible for muscle hypertonia. The coactivation of the leg muscles seen in children with cerebral palsy, which also is seen in the stepping of the newborn, suggests impaired maturation of the neuronal locomotor pattern.
针对痉挛型成年和儿童脑瘫患者在缓慢步态中踝关节运动以及腓肠肌产生的张力变化，分析了腿部肌肉的激活情况。在正常受试者中，步态站立期腓肠肌张力增加主要是由于腓肠肌和比目鱼肌肌电图增加。在痉挛型患者中，腓肠肌异常高张力的发展更多是由于被动肌肉拉伸，因为相互协调的腿部肌肉肌电图减少。得出的结论是，痉挛型患者的腿部伸肌由于其机械特性表现出假牵张反射行为，这是肌肉张力亢进的主要原因。脑瘫儿童中出现的腿部肌肉共同激活现象，在新生儿迈步时也可见，提示神经运动模式成熟受损。
