Schroeder D P, Wooley C F, Leier C V
Chest. 1978 Sep;74(3):314-7. doi: 10.1378/chest.74.3.314.
Electrophysiologic studies were performed on a 73-year-old man with swallowing-induced supraventricular tachycardia, in order to define the characteristics of this unique dysrhythmia in this patient. Swallowing reliably provoked an automatic atrial focus type of atrial tachycardia, which usually changed into an atrioventricular nodal reentrant tachycardia when a critical delay in atrioventricular nodal conduction (atrio-His interval greater than or equal to 340 msec) was achieved. The atrioventricular nodal reentrant form of tachycardia did not occur spontaneously. The ease of induction and the duration of the episodes of supraventricular tachycardia were facilitated with the intravenous administration of atropine and ouabain and were decreased with administration of procainamide hydrochloride.
对一名73岁吞咽诱发室上性心动过速的男性进行了电生理研究，以明确该患者这种独特心律失常的特征。吞咽可靠地诱发了自动心房灶型房性心动过速，当房室结传导出现临界延迟（房室希氏间期大于或等于340毫秒）时，这种心动过速通常会转变为房室结折返性心动过速。房室结折返性心动过速不会自发出现。静脉注射阿托品和哇巴因可促进室上性心动过速发作的诱发及持续时间，而静脉注射盐酸普鲁卡因胺则会使其降低。
