使用抗补体剂K-76单羧酸，用人或豚鼠补体成分制备EAC1,4b,2a,3b和EAC4b,3b的简单方法。 - Suppr

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超能文献

Simple methods for preparing EAC1,4b,2a,3b and EAC4b,3b with human or guinea pig complement components using an anticomplementary agent, K-76 monocarboxylic acid.

作者信息

Hong K, Kinoshita T, Inoue K

出版信息

J Immunol. 1981 Jul;127(1):109-14.

PMID:6909271

Abstract

摘要

相似文献

Simple methods for preparing EAC1,4b,2a,3b and EAC4b,3b with human or guinea pig complement components using an anticomplementary agent, K-76 monocarboxylic acid.使用抗补体剂K-76单羧酸，用人或豚鼠补体成分制备EAC1,4b,2a,3b和EAC4b,3b的简单方法。

J Immunol. 1981 Jul;127(1):109-14.

An anticomplementary agent, K-76 monocarboxylic acid: its site and mechanism of inhibition of the complement activation cascade.一种抗补体剂，K-76单羧酸：其对补体激活级联反应的抑制位点及机制

J Immunol. 1979 Jun;122(6):2418-23.

Inhibitory effect of K-76 monocarboxylic acid, an anticomplementary agent, on the C3b inactivator system.抗补体剂K-76单羧酸对C3b灭活剂系统的抑制作用。

J Immunol. 1981 Jul;127(1):104-8.

Flow cytometric assay for phagocytosis of human monocytes mediated via Fc gamma-receptors and complement receptor CR1 (CD35).通过Fcγ受体和补体受体CR1（CD35）介导的人单核细胞吞噬作用的流式细胞术检测

Cytometry. 1991;12(7):677-86. doi: 10.1002/cyto.990120712.

Dual function of insoluble polyanions. Formation of C1 and C42 sites in addition to activation of the alternative pathway of complement.不溶性聚阴离子的双重功能。除了激活补体替代途径外，还形成C1和C42位点。

Monogr Allergy. 1977;12:78-81.

A new activity of complement component C3: cell-bound C3b potentiates lysis of erythrocytes by C5b,6 and terminal components.补体成分C3的一种新活性：细胞结合的C3b增强C5b、6及末端成分对红细胞的溶解作用。

J Immunol. 1976 Sep;117(3):830-4.

[Preparation of lyophilized reagents for determining the functional activity of complement C1, C2, C3, C4 and C5 components].[用于测定补体C1、C2、C3、C4和C5成分功能活性的冻干试剂的制备]

Lab Delo. 1988(3):28-31.

Evidence for restriction of the ability of complement to lyse homologous erythrocytes.补体溶解同源红细胞能力受限的证据。

J Immunol. 1984 Sep;133(3):1444-52.

Complement receptors on guinea pig epidermal Langerhans cells.豚鼠表皮朗格汉斯细胞上的补体受体

J Immunol. 1980 Feb;124(2):685-90.

Hemolytic efficiency of cell-bound IgM: evidence that IgM-C1 complexes activate C4 molecules not hemolytic with homologous C2--C9.细胞结合型IgM的溶血效率：IgM-C1复合物激活不能与同源C2-C9发生溶血反应的C4分子的证据。

J Immunol. 1982 Apr;128(4):1600-2.

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Identification and partial characterization of the secreted form of the fourth component of human complement: evidence that it is different from major plasma form.人补体第四成分分泌形式的鉴定及部分特性分析：证明其与主要血浆形式不同。

Proc Natl Acad Sci U S A. 1983 Jan;80(1):268-72. doi: 10.1073/pnas.80.1.268.

Transmembrane channel formation by complement: functional analysis of the number of C5b6, C7, C8, and C9 molecules required for a single channel.补体形成跨膜通道：单个通道所需C5b6、C7、C8和C9分子数量的功能分析。

Proc Natl Acad Sci U S A. 1982 Aug;79(15):4751-5. doi: 10.1073/pnas.79.15.4751.

Identification of an additional class of C3-binding membrane proteins of human peripheral blood leukocytes and cell lines.鉴定人类外周血白细胞和细胞系中另一类C3结合膜蛋白。

Proc Natl Acad Sci U S A. 1985 Feb;82(3):859-63. doi: 10.1073/pnas.82.3.859.

Anti-peripheral myelin antibody in patients with demyelinating neuropathy: quantitative and kinetic determination of serum antibody by complement component 1 fixation.脱髓鞘性神经病患者的抗外周髓鞘抗体：通过补体成分1固定法对血清抗体进行定量和动力学测定。

Proc Natl Acad Sci U S A. 1985 Feb;82(3):905-9. doi: 10.1073/pnas.82.3.905.

Inhibition of the alternative C3 convertase and classical C5 convertase of complement by group A streptococcal M protein.A组链球菌M蛋白对补体替代途径C3转化酶和经典途径C5转化酶的抑制作用。

Infect Immun. 1990 Aug;58(8):2535-41. doi: 10.1128/iai.58.8.2535-2541.1990.

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