患有佝偻病的低出生体重婴儿的血浆活性维生素D浓度及其对维生素D治疗的反应。
Plasma active vitamin D concentration in low birthweight infants with rickets and its response to vitamin D treatment.
作者信息
Seino Y, Ishii T, Shimotsuji T, Ishida M, Yabuuchi H
出版信息
Arch Dis Child. 1981 Aug;56(8):628-32. doi: 10.1136/adc.56.8.628.
Abstract
Impairment of 25-hydroxylation may be a cause of rickets in infants of low birthweight, weighing between 2000 and 2500 g. In addition there may be impairment of 1 alpha-hydroxylation in infants weighing less than 2000 g. Our data show that a supplementary dose of vitamin D2 of at least 500 IU daily is a reasonable regimen for infants who weighed between 2000 and 2500 g at birth. However for infants who weighed less than 2000 g with a gestation of under 38 weeks, administration of 1 alpha-OHD3 may be more effective in preventing rickets.
摘要
25-羟化受损可能是出生体重在2000至2500克之间的低体重婴儿患佝偻病的一个原因。此外，出生体重低于2000克的婴儿可能存在1α-羟化受损。我们的数据表明，对于出生时体重在2000至2500克之间的婴儿，每天补充至少500国际单位的维生素D2是一种合理的方案。然而，对于出生体重低于2000克且孕周不足38周的婴儿，给予1α-羟化胆钙化醇在预防佝偻病方面可能更有效。
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Plasma active vitamin D concentration in low birthweight infants with rickets and its response to vitamin D treatment.患有佝偻病的低出生体重婴儿的血浆活性维生素D浓度及其对维生素D治疗的反应。
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