Heiss W D, Vyska K, Kloster G, Traupe H, Freundlieb C, Hoeck A, Feinendegen L E, Stoecklin G
Neuroradiology. 1982;23(1):45-7. doi: 10.1007/BF00399705.
A hemorrhagic infarction which caused left hemiparesis and homonymous hemianopia was demonstrated as a hypodense lesion on the first CT; 2 weeks later it was hyperdense and enhanced after injecting meglumine. Positron emission tomography with [11C]methyl-D-glucose also revealed the ischemic lesion, but also marked decrease of glucose uptake in the visual cortex, which indicated decreased functional activity.
首次CT显示为低密度灶的出血性梗死导致左侧偏瘫和同向性偏盲；2周后，该梗死灶呈高密度，注射葡甲胺后强化。[11C]甲基-D-葡萄糖正电子发射断层扫描也显示出缺血性病变，且视皮质葡萄糖摄取明显减少，提示功能活动降低。
