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Defective tumoricidal capacity of macrophages from P/J mice: tumoricidal defect involves abnormalities in lymphokine-derived activation stimuli and in mononuclear phagocyte responsiveness.

作者信息

Boraschi D, Meltzer M S

出版信息

J Immunol. 1980 Aug;125(2):777-82.

PMID:6993563
Abstract
摘要

相似文献

1
Defective tumoricidal capacity of macrophages from P/J mice: tumoricidal defect involves abnormalities in lymphokine-derived activation stimuli and in mononuclear phagocyte responsiveness.P/J小鼠巨噬细胞的肿瘤杀伤能力缺陷：肿瘤杀伤缺陷涉及淋巴因子衍生的激活刺激和单核吞噬细胞反应性的异常。
J Immunol. 1980 Aug;125(2):777-82.
2
Defective tumoricidal capacity of macrophages from P/J mice: characterization of the macrophage cytotoxic defect after in vivo and in vitro activation stimuli.P/J 小鼠巨噬细胞的肿瘤杀伤能力缺陷：体内和体外激活刺激后巨噬细胞细胞毒性缺陷的特征
J Immunol. 1980 Aug;125(2):771-6.
3
Defective tumoricidal capacity of macrophages from A/J mice. I. Characterization of the macrophage cytotoxic defect after in vivo and in vitro activation stimuli.A/J小鼠巨噬细胞的肿瘤杀伤能力缺陷。I. 体内和体外激活刺激后巨噬细胞细胞毒性缺陷的特征
J Immunol. 1979 Apr;122(4):1587-91.
4
Defective tumoricidal capacity of macrophages from A/J mice. II. Comparison of the macrophage cytotoxic defect of A/J mice with that of lipid A-unresponsive C3H/HeJ mice.A/J小鼠巨噬细胞的肿瘤杀伤能力缺陷。II. A/J小鼠巨噬细胞细胞毒性缺陷与脂质A无反应性C3H/HeJ小鼠的比较。
J Immunol. 1979 Apr;122(4):1592-97.
5
Macrophage activation for tumor cytotoxicity: induction of tumoricidal macrophages by supernatants of PPD-stimulated Bacillus Calmette-Guérin-immune spleen cell cultures.巨噬细胞激活以实现肿瘤细胞毒性：通过卡介苗刺激的卡介苗免疫脾细胞培养上清液诱导杀肿瘤巨噬细胞。
J Immunol. 1977 Sep;119(3):889-96.
6
Defective tumoricidal capacity of macrophages from A/J mice. III. Genetic analysis of the macrophage defect.
J Immunol. 1980 Mar;124(3):1050-3.
7
Similarities among factors that render macrophages tumoricidal in lymphokine and interferon preparations.淋巴因子和干扰素制剂中使巨噬细胞具有杀肿瘤细胞活性的各因素间的相似性。
Cancer Res. 1978 Apr;38(4):1003-7.
8
Induction, maintenance, and reinduction of tumoricidal activity in bone marrow-derived mononuclear phagocytes by Corynebacterium parvum. Evidence for the involvement of a T cell- and interferon-gamma-independent pathway of macrophage activation.短小棒状杆菌对骨髓来源的单核吞噬细胞杀肿瘤活性的诱导、维持及再诱导。巨噬细胞激活的T细胞和γ干扰素非依赖性途径参与的证据。
J Immunol. 1987 Apr 1;138(7):2366-71.
9
Macrophages in resistance to rickettsial infections: characterization of lymphokines that induce rickettsiacidal activity in macrophages.巨噬细胞对立克次氏体感染的抗性：诱导巨噬细胞产生杀立克次氏体活性的淋巴因子的特性
J Immunol. 1981 Jan;126(1):204-7.
10
Macrophages as cytotoxic effector cells.巨噬细胞作为细胞毒性效应细胞。
Immunobiology. 1982 Apr;161(3-4):401-7. doi: 10.1016/S0171-2985(82)80098-3.

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Phylogenetic variation and polymorphism at the toll-like receptor 4 locus (TLR4).Toll样受体4基因座（TLR4）的系统发育变异和多态性。
Genome Biol. 2000;1(1):RESEARCH002. doi: 10.1186/gb-2000-1-1-research002. Epub 2000 Apr 27.
2
Defective immunoglobulin M responses to vaccination or infection with Schistosoma mansoni in xid mice.xid小鼠对曼氏血吸虫疫苗接种或感染的免疫球蛋白M反应存在缺陷。
Infect Immun. 1985 Nov;50(2):409-14. doi: 10.1128/iai.50.2.409-414.1985.
3
Rapid killing of actinomycin D-treated tumour cells by mononuclear phagocytes: reactivity in mouse strains with defective classical tumour cytotoxicity.
单核吞噬细胞对放线菌素 D 处理的肿瘤细胞的快速杀伤作用：经典肿瘤细胞毒性存在缺陷的小鼠品系中的反应性
Immunology. 1986 Feb;57(2):249-53.
4
Mycobacterium bovis BCG-induced protection against cutaneous and systemic Leishmania major infections of mice.牛分枝杆菌卡介苗诱导的对小鼠皮肤和全身性利什曼原虫主要感染的保护作用。
Infect Immun. 1987 Jul;55(7):1707-14. doi: 10.1128/iai.55.7.1707-1714.1987.
5
Genetic complementation of defects in vaccine-induced immunity against Schistosoma mansoni in P- and A-strain inbred mice.P株和A株近交系小鼠中针对曼氏血吸虫疫苗诱导免疫缺陷的基因互补作用。
Infect Immun. 1988 Mar;56(3):708-10. doi: 10.1128/iai.56.3.708-710.1988.
6
Genetic association of defects in macrophage larvicidal activity and vaccine-induced resistance to Schistosoma mansoni in P strain mice.P品系小鼠巨噬细胞杀幼虫活性缺陷与疫苗诱导的曼氏血吸虫抗性的遗传关联。
Infect Immun. 1987 Aug;55(8):1884-9. doi: 10.1128/iai.55.8.1884-1889.1987.
7
Reassessment of the role of splenic leukocyte oxidative activity and macrophage activation in expression of immunity to malaria.重新评估脾脏白细胞氧化活性和巨噬细胞活化在疟疾免疫表达中的作用。
Infect Immun. 1989 Dec;57(12):3677-82. doi: 10.1128/iai.57.12.3677-3682.1989.
8
Lack of binding of bacterial lipopolysaccharide to mouse lung macrophages and restoration of binding by gamma interferon.细菌脂多糖与小鼠肺巨噬细胞的结合缺失以及γ干扰素对结合的恢复作用。
J Exp Med. 1985 Nov 1;162(5):1444-59. doi: 10.1084/jem.162.5.1444.