Hooper I, Egan J B
J Virol. 1981 Nov;40(2):599-601. doi: 10.1128/JVI.40.2.599-601.1981.
We show that coliphage 186 infection is dependent upon host initiation functions, dnaA and dnaC, which differentiates the phage from lambda and P2. The possibility is therefore entertained that the delay in 186 replication seen after infection of UV-irradiated bacterial cells reflects the temporary unavailability of one or both these functions. Infections with P1 and Mu need host dnaC but not dnaA and show some sensitivity to preirradiation of the host but are not as sensitive as 186.
我们发现大肠杆菌噬菌体186的感染依赖于宿主起始功能dnaA和dnaC,这使该噬菌体与λ噬菌体和P2噬菌体有所不同。因此,有人认为,在紫外线照射过的细菌细胞被感染后观察到的186复制延迟反映了这两种功能中一种或两种暂时无法使用。P1噬菌体和Mu噬菌体的感染需要宿主的dnaC,但不需要dnaA,并且对宿主的预照射有一定敏感性,但不如186敏感。