Esteban A, Gomez-Acebo E, de la Cal M A
Chest. 1982 Jan;81(1):47-50. doi: 10.1378/chest.81.1.47.
Sixteen patients, admitted no later than eight hours after the onset of the first stage of acute myocardial infarction, underwent systemic, pulmonary, and pulmonary wedge pressure measurements on admission and 24 and 48 hours after the first symptoms. A decrease in systolic blood pressure during inspiration equal to or greater than 15 mm Hg was found in 56 percent of the cases at admission, 53 percent at 24 hours, and 41 percent at 48 hours. No correlation could be found between this and: (1) pulmonary wedge pressure, (2) arteriovenous oxygen difference (volume percent), (3) location, and (4) extension of the necrosis. The last was indirectly assessed by the maximum levels of creatine phosphokinase attained. We discuss the previously postulated mechanisms causing pulsus paradoxus and formulate a theory to explain in acute uncomplicated myocardial infarction.
16例在急性心肌梗死第一阶段发作后8小时内入院的患者，在入院时以及出现首发症状后24小时和48小时接受了全身、肺和肺楔压测量。入院时56%的病例、24小时时53%的病例以及48小时时41%的病例发现吸气时收缩压下降等于或大于15 mmHg。在这与以下各项之间未发现相关性：(1)肺楔压，(2)动静脉氧差（容积百分比），(3)坏死部位，(4)坏死范围。后者通过肌酸磷酸激酶达到的最高水平间接评估。我们讨论了先前假定的导致奇脉的机制，并提出了一种理论来解释急性非复杂性心肌梗死中的奇脉现象。
