Roze L V, Rozenfel'd E L
Vopr Med Khim. 1978 Sep-Oct;24(5):690-4.
Adrenaline, poradrenaline, serotonin, triptamine and 3-hydroxytyramine activated glycogen phosphorolysis in heart of nonanesthetized rats due to increase in the phosphorylase A activity. Anesthesia with nembutal and ether prevented the stimulating effect of biogenic amines (excluding serotonin) on phosphorolysis. Adrenaline, administered into animals anesthetized with nembutal, inhibited the glycogen phosphorolysis. Noradrenaline caused a decrease in gamma-amylolysis of glycogen in anesthetized and untreated rats. The inhibitory effect of adrenaline on glycogen gamma-amylolysis occurred in rat heart muscle only under the conditions of anesthesia.
肾上腺素、去甲肾上腺素、血清素、色胺和3-羟酪胺可激活未麻醉大鼠心脏中的糖原磷酸化作用，原因是磷酸化酶A的活性增加。戊巴比妥和乙醚麻醉可阻止生物胺（血清素除外）对磷酸化作用的刺激效果。给用戊巴比妥麻醉的动物注射肾上腺素，会抑制糖原磷酸化作用。去甲肾上腺素可使麻醉和未处理大鼠的糖原γ-淀粉酶解作用降低。肾上腺素对糖原γ-淀粉酶解作用的抑制效果仅在麻醉条件下的大鼠心肌中出现。
