Langeron P, Harle J, Robert J L
Phlebologie. 1982 Jan-Mar;35(1):273-87.
Venous stasis in post-phlebitic syndromes may be caused by several physiopathological processes. Phlebography and functional investigations (Doppler, occlusive rheoplethysmography) enables us to make a fairly precise analysis of the causal factors: --The obstruction of a large venous collector defines the obstructive syndromes. The size of the stasis depends on the functional value of the substitutional channels; distinction may be made between compensated obstructive syndromes (with efficient substitutional circulation) and poorly or non-compensated obstructive syndromes. --Orthostatic reflux in devalvulated deep veins constitutes another factor of stasis. Distinction to be made between minor reflux, and major reflux with massive reflux in the great trunks and associated insufficiency of the tibial communicators. --Post-phlebitic superficial venous insufficiency is often observed. Its physiopathology is basically no different to that of primary varices, the functioning of the tibial communicators is often perturbed. --The restrictive syndrome, result of functional investigations, corresponds to a reduction of the venous distensibility whose effects are superadded to those of the syndromes described above.
静脉炎后综合征中的静脉淤滞可能由多种生理病理过程引起。静脉造影和功能检查（多普勒、阻塞性血流容积描记法）使我们能够对病因进行相当精确的分析：——大静脉收集器的阻塞定义了阻塞性综合征。淤滞的程度取决于替代通道的功能状态；可区分代偿性阻塞综合征（具有有效的替代循环）和代偿不良或无代偿的阻塞综合征。——瓣膜功能不全的深静脉中的直立性反流是另一个淤滞因素。需区分轻度反流和大静脉主干大量反流及相关胫部交通支功能不全的重度反流。——常观察到静脉炎后浅静脉功能不全。其生理病理与原发性静脉曲张基本无异，胫部交通支的功能常受干扰。——功能检查结果显示的限制性综合征，对应静脉扩张性降低，其影响叠加于上述综合征的影响之上。
