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The hepatic toxicity of isoniazid among rapid and slow acetylators of the drug.

作者信息

Ellard G A, Mitchison D A, Girling D J, Nunn A J, Fox W

出版信息

Am Rev Respir Dis. 1978 Sep;118(3):628-9. doi: 10.1164/arrd.1978.118.3.628.

DOI:10.1164/arrd.1978.118.3.628
PMID:707886
Abstract
摘要

相似文献

1
The hepatic toxicity of isoniazid among rapid and slow acetylators of the drug.
Am Rev Respir Dis. 1978 Sep;118(3):628-9. doi: 10.1164/arrd.1978.118.3.628.
2
Predisposing factors in hepatitis induced by isoniazid-rifampin treatment of tuberculosis.异烟肼-利福平治疗结核病所致肝炎的诱发因素。
Am Rev Respir Dis. 1978 Sep;118(3):461-6. doi: 10.1164/arrd.1978.118.3.461.
3
Rifampin-induced release of hydrazine from isoniazid. A possible cause of hepatitis during treatment of tuberculosis with regimens containing isoniazid and rifampin.利福平诱导异烟肼释放肼。含异烟肼和利福平方案治疗结核病期间肝炎的一个可能原因。
Am Rev Respir Dis. 1986 Jun;133(6):1072-5. doi: 10.1164/arrd.1986.133.6.1072.
4
[The potential clinical significance of the isoniazid acetylator phenotype in the treatment of pulmonary tuberculosis].[异烟肼乙酰化酶表型在肺结核治疗中的潜在临床意义]
Rev Mal Respir. 1984;1(4):207-19.
5
Lack of relationship between hepatic toxicity and acetylator phenotype in three thousand South Indian patients during treatment with isoniazid for tuberculosis.三千名南印度患者在接受异烟肼治疗结核病期间,肝毒性与乙酰化代谢表型之间不存在相关性。
Am Rev Respir Dis. 1984 Jan;129(1):58-61. doi: 10.1164/arrd.1984.129.1.58.
6
NAT2 genotype guided regimen reduces isoniazid-induced liver injury and early treatment failure in the 6-month four-drug standard treatment of tuberculosis: a randomized controlled trial for pharmacogenetics-based therapy.NAT2 基因型指导方案可降低利福平肝损伤和 6 个月四联标准抗结核治疗早期治疗失败率:一项基于遗传药理学的治疗的随机对照试验。
Eur J Clin Pharmacol. 2013 May;69(5):1091-101. doi: 10.1007/s00228-012-1429-9. Epub 2012 Nov 14.
7
Increased incidence of isoniazid hepatitis in rapid acetylators: possible relation to hydranize metabolites.快乙酰化者中异烟肼肝炎发病率增加:可能与肼类代谢产物有关。
Clin Pharmacol Ther. 1975 Jul;18(1):70-9. doi: 10.1002/cpt197518170.
8
Determining the relation between N-acetyltransferase-2 acetylator phenotype and antituberculosis drug induced hepatitis by molecular biologic tests.通过分子生物学检测确定N-乙酰转移酶-2乙酰化表型与抗结核药物性肝炎之间的关系。
Tuberk Toraks. 2008;56(1):81-6.
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The hepatic toxicity of antituberculosis regimens containing isoniazid, rifampicin and pyrazinamide.含异烟肼、利福平及吡嗪酰胺的抗结核治疗方案的肝毒性
Tubercle. 1978 Mar;59(1):13-32. doi: 10.1016/0041-3879(77)90022-8.
10
Hepatic toxicity in South Indian patients during treatment of tuberculosis with short-course regimens containing isoniazid, rifampicin and pyrazinamide.
Tubercle. 1986 Jun;67(2):99-108. doi: 10.1016/0041-3879(86)90003-6.

引用本文的文献

1
Involvement of endoplasmic reticulum stress in rifampicin-induced liver injury.内质网应激在利福平诱导的肝损伤中的作用
Front Pharmacol. 2022 Oct 20;13:1022809. doi: 10.3389/fphar.2022.1022809. eCollection 2022.
2
Protective effects of metallothionein on isoniazid and rifampicin-induced hepatotoxicity in mice.金属硫蛋白对异烟肼和利福平诱导的小鼠肝毒性的保护作用。
PLoS One. 2013 Aug 13;8(8):e72058. doi: 10.1371/journal.pone.0072058. eCollection 2013.
3
Arylamine N-acetyltransferases: a structural perspective.芳香胺 N-乙酰基转移酶:结构视角。
Br J Pharmacol. 2013 Jun;169(4):748-60. doi: 10.1111/bph.12182.
4
Drug-induced hepatic disorders. Incidence, management and avoidance.药物性肝病。发病率、管理与预防
Drug Saf. 1993 Dec;9(6):441-9. doi: 10.2165/00002018-199309060-00007.
5
Late effects following isoniazid therapy.异烟肼治疗后的迟发效应。
Am J Public Health. 1980 Sep;70(9):987-9. doi: 10.2105/ajph.70.9.987.
6
Hydrallazine-induced hepatitis?肼屈嗪诱发的肝炎?
Br Med J. 1980 May 10;280(6224):1165-6. doi: 10.1136/bmj.280.6224.1165.
7
A genetic component of the variance of N-acetoxy-2-acetylaminofluorene-induced DNA damage in mononuclear leukocytes determined by a twin study.一项双胞胎研究确定的N-乙酰氧基-2-乙酰氨基芴诱导的单核白细胞DNA损伤变异中的遗传成分。
Hum Genet. 1983;65(2):181-4. doi: 10.1007/BF00286659.
8
Adverse effects of antituberculosis drugs.抗结核药物的不良反应。
Drugs. 1982 Jan-Feb;23(1-2):56-74. doi: 10.2165/00003495-198223010-00003.
9
Antituberculous therapy in children.儿童抗结核治疗
Indian J Pediatr. 1986 Mar-Apr;53(2):179-98. doi: 10.1007/BF02748507.
10
Genetically determined variability in acetylation and oxidation. Therapeutic implications.乙酰化和氧化的基因决定变异性。治疗意义。
Drugs. 1985 Apr;29(4):342-75. doi: 10.2165/00003495-198529040-00003.