Brizzi E, Calamai F, Staderini G
Boll Soc Ital Biol Sper. 1982 Mar 30;58(6):300-5.
Electrical vagal nerve stimulation (10-20 V, 0.2-5 msec, 1-86 Hz) was carried out at cervical level in nembutal-urethane anestetized rabbits. The aim of the research was to selectively activate (0.2 msec) "low" excitatory, and (5 msec) "high" inhibitory efferent vagal nerve fibers supplying the stomach. In the intact stomach an increasing motor effect was evoked between 1 and 16 Hz. An inhibitory motor effect became, instead, evident at higher frequencies of stimulation (16-86 Hz) In atropinized animals excitatory motor effects were blocked. The inhibitory effects were instead still present and they were not affected by guanethidine. These results suggest a non-cholinergic, non-adrenergic mechanism for the vagal inhibitory control of gastric motility in rabbit.
在戊巴比妥-乌拉坦麻醉的家兔颈部进行电刺激迷走神经（10 - 20伏，0.2 - 5毫秒，1 - 86赫兹）。该研究的目的是选择性激活（0.2毫秒）“低”兴奋性和（5毫秒）“高”抑制性传出迷走神经纤维，这些纤维支配胃。在完整的胃中，1至16赫兹之间可诱发运动效应增强。相反，在较高刺激频率（16 - 86赫兹）时，抑制性运动效应变得明显。在阿托品化的动物中，兴奋性运动效应被阻断。相反，抑制性效应仍然存在，且不受胍乙啶影响。这些结果提示家兔胃运动的迷走神经抑制性控制存在非胆碱能、非肾上腺素能机制。
