Palmer K R, Jenkins W J
Gut. 1982 Sep;23(9):729-33. doi: 10.1136/gut.23.9.729.
High blood acetaldehyde levels in alcoholics after ethanol ingestion are due to reduced acetaldehyde oxidation rather than to an increased rate of its formation from ethanol. This is associated with low hepatic acetaldehyde dehydrogenase activity in alcoholic subjects and may represent a specific abnormality in them.
酒精摄入后,酗酒者血液中乙醛水平升高是由于乙醛氧化减少,而非乙醇生成乙醛的速率增加。这与酗酒者肝脏中乙醛脱氢酶活性较低有关,可能是他们特有的一种异常情况。