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Post-injury myelin-breakdown products inhibit axonal growth: an hypothesis to explain the failure of axonal regeneration in the mammalian central nervous system.

作者信息

Berry M

出版信息

Bibl Anat. 1982(23):1-11.

PMID:7138484
Abstract
摘要

相似文献

1
Post-injury myelin-breakdown products inhibit axonal growth: an hypothesis to explain the failure of axonal regeneration in the mammalian central nervous system.损伤后髓磷脂分解产物抑制轴突生长：一种解释哺乳动物中枢神经系统轴突再生失败的假说。
Bibl Anat. 1982(23):1-11.
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Contact inhibition in the failure of mammalian CNS axonal regeneration.接触抑制与哺乳动物中枢神经系统轴突再生失败
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Myelin from peripheral and central nervous system is a nonpermissive substrate for retinal ganglion cell axons.来自外周和中枢神经系统的髓磷脂是视网膜神经节细胞轴突的非允许性底物。
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Nerve fibre regeneration after traumatic lesions of the CNS; progress and problems.
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Laminin overrides the inhibitory effects of peripheral nervous system and central nervous system myelin-derived inhibitors of neurite growth.层粘连蛋白可克服外周神经系统和中枢神经系统髓磷脂源性神经突生长抑制因子的抑制作用。
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A review focuses on a neglected and controversial component of SCI: myelin debris.一篇综述聚焦于脊髓损伤（SCI）中一个被忽视且存在争议的成分：髓磷脂碎片。
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