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Protection from irreversible hypoxic injury by potassium cardioplegia and hypothermia: effects on contracture, morphology and O2-enzyme release.

作者信息

Ganote C E, Angelo J, Safavi S, Kaltenbach J P

出版信息

J Mol Cell Cardiol. 1982 Oct;14(10):587-99. doi: 10.1016/0022-2828(82)90145-6.

DOI:10.1016/0022-2828(82)90145-6
PMID:7154096
Abstract
摘要

相似文献

1
Protection from irreversible hypoxic injury by potassium cardioplegia and hypothermia: effects on contracture, morphology and O2-enzyme release.
J Mol Cell Cardiol. 1982 Oct;14(10):587-99. doi: 10.1016/0022-2828(82)90145-6.
2
Oxygen-induced enzyme release after irreversible myocardial injury. Effects of cyanide in perfused rat hearts.不可逆性心肌损伤后氧诱导的酶释放。氰化物对灌注大鼠心脏的影响。
Am J Pathol. 1976 Aug;84(2):327-50.
3
Modification of caffeine-induced injury in Ca2+-free perfused rat hearts. Relationship to the calcium paradox.无钙灌注大鼠心脏中咖啡因诱导损伤的改变。与钙反常的关系。
Am J Pathol. 1986 May;123(2):351-64.
4
Does potassium potentiate profound hypothermic cardioplegia for myocardial preservation?钾是否能增强深度低温停搏液对心肌的保护作用?
Surgery. 1978 Jul;84(1):94-103.
5
Hypothermic potassium cardioplegia preserves myocardial compliance.低温钾停搏液可维持心肌顺应性。
Surgery. 1979 Dec;86(6):810-7.
6
The additive protective effects of hypothermia and chemical cardioplegia during ischemic cardiac arrest in the rat.
J Thorac Cardiovasc Surg. 1980 Jan;79(1):39-43.
7
[Myocardial metabolism during coronary perfusion at 10 degrees C with or without cardioplegia associated with potassium].10℃下冠状动脉灌注时,伴有或不伴有钾相关心脏停搏液的心肌代谢
Arch Mal Coeur Vaiss. 1980 Sep;73(9):1075-85.
8
Parallel temperature dependence of contracture-associated enzyme release due to anoxia, 2,4-dinitrophenol (DNP), or caffeine and the calcium paradox.缺氧、2,4-二硝基苯酚(DNP)或咖啡因引起的挛缩相关酶释放与钙反常的平行温度依赖性。
Am J Pathol. 1984 Jul;116(1):94-106.
9
Cold-blood potassium cardioplegia: evaluation of glutathione and postischemic cardioplegia.
J Thorac Cardiovasc Surg. 1979 Dec;78(6):893-907.
10
Advantages of potassium cardioplegia and perfusion hypothermia in left ventricular hypertrophy.钾离子停搏液与灌注性低温对左心室肥厚的优势
Ann Thorac Surg. 1977 Oct;24(4):229-306. doi: 10.1016/s0003-4975(10)63404-2.

引用本文的文献

1
Ultrastructure of cultured adult myocardial cells during anoxia and reoxygenation.缺氧和复氧过程中培养的成年心肌细胞的超微结构
Am J Pathol. 1984 Jun;115(3):349-61.
2
Absence of reoxygenation damage in isolated heart cells after anoxic injury.缺氧损伤后分离的心脏细胞中无再氧合损伤。
Pflugers Arch. 1984 May;401(1):71-6. doi: 10.1007/BF00581535.
3
Structural changes in cardiac gap junctions after hypoxia and reoxygenation: a quantitative freeze-fracture analysis.缺氧和复氧后心脏缝隙连接的结构变化:定量冷冻断裂分析
Cell Tissue Res. 1990 Jul;261(1):183-94. doi: 10.1007/BF00329451.