Batory I
Z Orthop Ihre Grenzgeb. 1982 Nov-Dec;120(6):833-49. doi: 10.1055/s-2008-1051405.
Due to the varying development of the vasculature of the proximal end of the femur, with a diminishing number of arteries ensuring blood supply and the subsequent reduction in the speed of flow in the myeloic sinuses, we describe here the origin of the latent ischemic phase of the proximal epiphysis of the femur, which constitutes the predisposition for the development of Perthes' disease. The hypoplasia of the arteries ensuring the blood supply of the proximal epiphysis of the femur may result in a latent ischemic phase which, under the influence of trigger factors, such as inflammations, traumas, etc., may lead to the evolution of Perthes' disease. The latent ischemic phase implies a phase of reduced resistance of the nucleus of ossification of the proximal epiphysis of the femur and corresponds to the "first infarct" in the findings of Inoue, Freeman and collaborators (1976). At the same time, it explains the findings of these and other authors in the sense that the "first infarct" affects the entire epiphyseal nucleus of the bone, a phenomenon which was not sufficiently explained by means of the theory of repeated infarcts. This publication is based on our own findings based on sections and animal experiments.
由于股骨近端血管系统发育各异，供应血液的动脉数量减少，随后骨髓窦内血流速度降低，我们在此描述股骨近端骨骺潜在缺血期的起源，这构成了佩特兹病发展的易患因素。确保股骨近端骨骺血液供应的动脉发育不全可能导致潜在缺血期，在炎症、创伤等触发因素的影响下，可能会引发佩特兹病的演变。潜在缺血期意味着股骨近端骨骺骨化核抵抗力降低的阶段，与井上、弗里曼及其合作者（1976年）研究结果中的“首次梗死”相对应。同时，从“首次梗死”影响整个骨骺骨核这一意义上解释了这些作者及其他作者的研究结果，而重复梗死理论对此现象的解释并不充分。本出版物基于我们自己通过切片和动物实验得出的研究结果。
