Batory I
Arch Orthop Trauma Surg (1978). 1982;100(3):151-62. doi: 10.1007/BF00442727.
The article describes the various opinions on the etiology of Legg-Calvé-Perthes disease: 1. The theory of double infarction. 2. The theory of venous stasis. 3. The latent prolonged ischemic phase. Due to the varying development of the vasculature of the proximal end of the femur, with a diminishing number of arteries ensuring blood supply and the subsequent reduction in the speed of flow in the myeloic sinuses, we describe here the origin of the latent ischemic phase of the proximal epiphysis of the femur, which constitutes the predisposition for the development of Perthes' disease. In the discussion about the diverse alterations connected with Legg-Calvé-Perthes disease, like the retardation of skeletal maturity, the dysplasia capitis or the necrosis following the treatment of congenital hip luxation, the different interpretations about the etiology of Perthes' disease are compared.
本文描述了关于Legg-Calvé-Perthes病病因的各种观点:1. 双梗死理论。2. 静脉淤滞理论。3. 潜在的长期缺血期。由于股骨近端血管系统发育各异,供应血液的动脉数量减少,随后骨髓窦内血流速度降低,我们在此描述股骨近端骨骺潜在缺血期的起源,这是佩特兹病发病的易患因素。在关于与Legg-Calvé-Perthes病相关的各种改变(如骨骼成熟延迟、股骨头发育异常或先天性髋关节脱位治疗后的坏死)的讨论中,对佩特兹病病因的不同解释进行了比较。