Takashima S, Armstrong D, Becker L, Bryan C
Ann Neurol. 1978 Sep;4(3):257-62. doi: 10.1002/ana.410040312.
Gliosis is increased in the respiratory control area of the brainstem in victims of sudden infant death syndrome (SIDS), as it is in infants who have died of congenital heart disease. In the latter, the lesions appear to result from hypoxia or ischemia, and studies of the brainstem microvasculature of SIDS victims indicated a close relationship between the gliosis and adjacent vasculature. It is postulated that cerebral hypoperfusion may play a role in SIDS.
与死于先天性心脏病的婴儿一样，婴儿猝死综合征（SIDS）受害者脑干呼吸控制区域的神经胶质增生也会增加。对于死于先天性心脏病的婴儿，这些病变似乎是由缺氧或局部缺血引起的，而对SIDS受害者脑干微血管系统的研究表明，神经胶质增生与相邻的血管系统之间存在密切关系。据推测，脑灌注不足可能在SIDS中起作用。
