肿瘤源性抑制因子对巨噬细胞吞噬金黄色葡萄球菌的作用
Suppressive factor of tumour origin against macrophage phagocytosis of Staphylococcus aureus.
作者信息
Saito H, Tomioka H
出版信息
Br J Cancer. 1980 Feb;41(2):259-67. doi: 10.1038/bjc.1980.38.
Abstract
Peritoneal macrophages from Sarcoma-180-bearing mice against Staphylococcus aureus were studied to determine the in vitro phagocytic capacities. When the phagocytic system was opsonized with normal mouse serum, macrophage phagocytic activity increased markedly soon after tumour graft and then returned to normal. A new antiphagocytic factor was detected in the serum of tumour-bearers soon after tumour implantation. This factor was of tumour origin, stable even at a temperature of 56 degrees C for 30 min and non-dialysable. Peptone-starch-induced macrophages were less sensitive to the factor than their unstimulated counterpart.
摘要
对荷肉瘤180小鼠的腹膜巨噬细胞针对金黄色葡萄球菌的体外吞噬能力进行了研究。当吞噬系统用正常小鼠血清调理后，肿瘤移植后不久巨噬细胞吞噬活性显著增加，然后恢复正常。肿瘤植入后不久在荷瘤小鼠血清中检测到一种新的抗吞噬因子。该因子起源于肿瘤，即使在56℃温度下30分钟仍稳定且不可透析。蛋白胨-淀粉诱导的巨噬细胞对该因子的敏感性低于未受刺激的巨噬细胞。
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本文引用的文献
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Subversion of host defense mechanisms by murine tumors. II. Counter-influence of concomitant antitumor immunity.小鼠肿瘤对宿主防御机制的颠覆。II. 伴随抗肿瘤免疫的反作用。
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Subversion of host defense mechanisms by murine tumors. I. A circulating factor that suppresses macrophage-mediated resistance to infection.小鼠肿瘤对宿主防御机制的破坏。I. 一种抑制巨噬细胞介导的抗感染抵抗力的循环因子。
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