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On the mechanism of the glutathione depletion induced in the liver by acute ethanol ingestion.

作者信息

Videla L A, Fernández V, Fernández N, Valenzuela A

出版信息

Subst Alcohol Actions Misuse. 1981;2(3):153-60.

PMID:7198294
Abstract
摘要

相似文献

1
On the mechanism of the glutathione depletion induced in the liver by acute ethanol ingestion.关于急性摄入乙醇引起肝脏中谷胱甘肽耗竭的机制。
Subst Alcohol Actions Misuse. 1981;2(3):153-60.
2
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Free radicals and not acetaldehyde influence the circulating levels of glutathione after acute or chronic alcohol abuse: in vivo and in vitro studies.自由基而非乙醛影响急性或慢性酒精滥用后谷胱甘肽的循环水平:体内和体外研究。
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引用本文的文献

1
Time course study of the changes in blood glutathione induced by acute ethanol intoxication in the rat.大鼠急性乙醇中毒所致血液中谷胱甘肽变化的时间进程研究
Experientia. 1983 Aug 15;39(8):880-2. doi: 10.1007/BF01990416.
2
Assessment of the scavenging action of reduced glutathione, (+)-cyanidanol-3 and ethanol by the chemiluminescent response of the xanthine oxidase reaction.通过黄嘌呤氧化酶反应的化学发光响应评估还原型谷胱甘肽、(+)-花青素-3和乙醇的清除作用。
Experientia. 1983 May 15;39(5):500-2. doi: 10.1007/BF01965175.
3
Conjugation of acetaldehyde with cysteinylglycine, the first metabolite in glutathione breakdown by gamma-glutamyltranspeptidase.
乙醛与半胱氨酰甘氨酸的结合,这是γ-谷氨酰转肽酶分解谷胱甘肽过程中的首个代谢产物。
Agents Actions. 1985 Oct;17(1):48-52. doi: 10.1007/BF01966681.