Love G J, Lan S p, Shy C M, Struba R J
Arch Environ Health. 1981 Mar-Apr;36(2):66-74. doi: 10.1080/00039896.1981.10667609.
The incidence and severity of acute respiratory disease was studied in families in three New York communities with different ambient levels of SO2 and particulate air pollution. Upper, lower, and total respiratory disease rates in fathers, mothers, and school children tended to be higher in the communities with higher pollution levels. Similar higher rates, however, were not observed among preschool children. Regression analyses were used to adjust rates for socioeconomic status, parental smoking, chronic bronchitis in parents, and possible indoor pollution resulting from the use of a gas stove for cooking. After these adjustments the community differences were still significant (P less than .01), for schoolchildren. The indoor pollution related to gas stoves was a significant covariate among children. The effects of smoking were inconsistent. It was not possible to attribute the higher rates observed to any specific pollutant, since both SO2 and particulate matter levels were higher in the high pollution communities, nor was it possible to attribute the excesses to current levels of exposure or to a residual effect of previous higher exposure concentrations. The fact that young children did not follow the pattern suggests the latter. It was concluded, however, that current or previous exposures to the complexity of air pollutants in New York City was at least partially responsible for increased incidences of acute respiratory disease.
在纽约三个社区中,研究了家庭中急性呼吸道疾病的发病率和严重程度,这些社区的二氧化硫和颗粒物空气污染水平各不相同。在污染水平较高的社区中,父亲、母亲和学龄儿童的上呼吸道、下呼吸道及总呼吸道疾病发病率往往更高。然而,在学龄前儿童中未观察到类似的较高发病率。采用回归分析对社会经济地位、父母吸烟情况、父母的慢性支气管炎以及因使用燃气灶具做饭可能导致的室内污染进行了发病率调整。在进行这些调整之后,学龄儿童的社区差异仍然显著(P小于0.01)。与燃气灶具相关的室内污染是儿童中的一个显著协变量。吸烟的影响并不一致。由于高污染社区中的二氧化硫和颗粒物水平均较高,因此无法将观察到的较高发病率归因于任何特定污染物,也无法将这些超额发病率归因于当前的暴露水平或先前较高暴露浓度的残留影响。幼儿未呈现这种模式这一事实表明是后者。然而,得出的结论是,纽约市目前或过去接触的复杂空气污染物至少部分导致了急性呼吸道疾病发病率的增加。