Carroll W, Stempel D, Lewiston N J
Ann Allergy. 1981 May;46(5):246-8.
Seventeen of 67 children recovering from status asthmaticus developed sleeping bradycardia. They were compared to a control population of asthmatic children who did not develop sleeping bradycardia and differed only in the low heart rate. The authors speculate that this phenomenon is due to a combination of withdrawal of sympathomimetic stimulation and catch-up in REM (rapid eye movement) sleep.
67名从哮喘持续状态中恢复的儿童中有17名出现睡眠性心动过缓。他们与未出现睡眠性心动过缓的哮喘儿童对照组进行了比较，两组仅在心率较低方面存在差异。作者推测，这种现象是由于拟交感神经刺激的消退和快速眼动睡眠的补偿作用共同导致的。
