肾脏镉过载但无肾毒性。
Renal cadmium overload without nephrotoxicity.
作者信息
Ghose R R, Morgan W D, Cummins P E
出版信息
Br J Ind Med. 1981 May;38(2):185-6. doi: 10.1136/oem.38.2.185.
Abstract
A redundant nickel/cadmium battery worker was investigated for non-specific fatigue after completing five years in the industry. Sensitive techniques for in-vivo organ cadmium measurement showed a moderate accumulation in the liver but a very large concentration in the kidneys. Despite this, overall glomerular and tubular function were not impaired. It was concluded that the mechanism of proteinuria observed in some cadmium workers is obscure and not clearly related to the degree of kidney saturation with cadmium.
摘要
一名从事镍镉电池工作五年的多余工人，因非特异性疲劳接受调查。体内器官镉测量的敏感技术显示，肝脏中有中度镉积累，但肾脏中的镉浓度非常高。尽管如此，总体肾小球和肾小管功能并未受损。得出的结论是，一些镉作业工人中观察到的蛋白尿机制尚不清楚，与肾脏镉饱和程度没有明显关系。
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