Sepúlveda F C, Duro L A, da Silva M M, Leite S R
Arq Neuropsiquiatr. 1981 Mar;39(1):106-14. doi: 10.1590/s0004-282x1981000100012.
The case of one 23 year-old girl who had epileptic manifestations is reported. At first, generalized tonic seizures; afterwards, epileptic seizures precipitated by eating. The electroencephalograms showed left temporal lobe dysfunctions. Different types of drugs were used with no success. The best results were obtained by association of sodium valproate, clonazepam and phenobarbital. Comments are made about clinic and etiopathogenesis, believing the authors in the hypothesis of nervous structures chronic hyperactivity. To Walker the hyperactivity was reached by hormones production under neural control of specific cerebral centers. The continuous bombardment of epileptic discharges to hypothalamic centers is the probably responsible by epileptic seizures precipitated by eating.
报告了一名23岁有癫痫表现女孩的病例。起初为全身性强直发作;之后,进食可诱发癫痫发作。脑电图显示左颞叶功能障碍。使用了不同类型的药物但均未成功。丙戊酸钠、氯硝西泮和苯巴比妥联合使用取得了最佳效果。对临床和病因发病机制进行了评论,作者相信神经结构慢性多动的假说。沃克认为,在特定脑中枢的神经控制下,激素产生导致了多动。癫痫放电对下丘脑中枢的持续轰击可能是进食诱发癫痫发作的原因。