Kul'chitskiĭ K I, Andreenko T V, Stechenko L A, Khvorostianaia T T
Arkh Anat Gistol Embriol. 1981 Oct;81(11):53-61.
Dynamics of decompensatory changes in the heart (in cardiomyocytes, connective tissue stroma of the myocardium, neural structures, microcirculatory bed) has been followed under conditions of developing stenosis of the pulmonary trunk. The data have been obtained by means of various investigational methods: histological - injection of vessels with Indian ink - gelatin, impregnation of the neural structures after Bielschowsky - Gros; histochemical - to reveal functional activity of parasympathetic and sympathetic neural structures after Koelle - Gomori and Falck - Hillarp - Govyrin, and electronmicroscopically. At the beginning of the experiment destructive processes predominate. By the first month, compensatory-adaptive processes develop in the heart. By the first year, persisting hypoxia and quick exhaustion of all cardiac structures result in development of the heart failure.
在肺动脉干狭窄发展的情况下，对心脏（心肌细胞、心肌结缔组织基质、神经结构、微循环床）失代偿性变化的动态进行了跟踪研究。数据通过各种研究方法获得：组织学方法——用印度墨水-明胶注射血管，采用 Bielschowsky - Gros 法对神经结构进行浸染；组织化学方法——采用 Koelle - Gomori 法和 Falck - Hillarp - Govyrin 法揭示副交感神经和交感神经结构的功能活性，以及电子显微镜观察法。实验开始时，破坏过程占主导。到第一个月时，心脏中出现代偿适应性过程。到第一年时，持续的缺氧以及所有心脏结构的快速耗竭导致心力衰竭的发生。
