用于产生细胞毒性反应中“信号2”的无细胞因子替代物。 - Suppr

Suppr

超能文献

Cell-free factor substitute for "signal 2" in generating cytotoxic reactions.

作者信息

Sopori M L, Alter B J, Bach F H, Bach M L

出版信息

Eur J Immunol. 1977 Nov;7(11):823-5. doi: 10.1002/eji.1830071115.

DOI:10.1002/eji.1830071115

PMID:73468

Abstract

摘要

相似文献

Cell-free factor substitute for "signal 2" in generating cytotoxic reactions.用于产生细胞毒性反应中“信号2”的无细胞因子替代物。

Eur J Immunol. 1977 Nov;7(11):823-5. doi: 10.1002/eji.1830071115.

Activation of secondary cytotoxic lymphocytes by cell-free factors from I-region-primed and D-region-primed lymphocytes.来自I区致敏淋巴细胞和D区致敏淋巴细胞的无细胞因子对二级细胞毒性淋巴细胞的激活作用。

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The antigen-specific signal in T-lymphocyte activation by alloantigen.同种异体抗原激活T淋巴细胞过程中的抗原特异性信号。

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Recruitment of functional subpopulations of T lymphocytes. Initiator T lymphocytes recruit helper T cells for cytotoxic response.T淋巴细胞功能亚群的募集。起始T淋巴细胞募集辅助性T细胞以进行细胞毒性反应。

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Relationships among differentiated T-cell subpopulations. II. Cytotoxicity and other functions of T cells specific for nucleated chicken erythrocytes.分化的T细胞亚群之间的关系。II. 对有核鸡红细胞具有特异性的T细胞的细胞毒性及其他功能。

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T-T cell interactions during in vitro cytotoxic T lymphocyte responses. III. Antigen-specific T helper cells release nonspecific mediator(s) able to help induction of H-2-restricted cytotoxic T lymphocyte responses across cell-impermeable membranes.体外细胞毒性T淋巴细胞反应过程中的T-T细胞相互作用。III. 抗原特异性T辅助细胞释放非特异性介质，能够帮助诱导跨细胞不可渗透膜的H-2限制性细胞毒性T淋巴细胞反应。

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Inhibition of cytotoxic T-cell clonal expansion by cyclophosphamide and the recovery of cytotoxic T-lymphocyte precursors by supernatants from mixed-lymphocyte cultures.

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Antigen-specific suppression of cytotoxic T cell responses in mice. I. Suppressor T cells are not cytotoxic cells.

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Major transplantation antigens in T cell-mediated immunity: a comparison of the transplantation reaction with antiviral immunity.T细胞介导免疫中的主要移植抗原：移植反应与抗病毒免疫的比较。

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[Cytoxicity of T lymphocytes].[T淋巴细胞的细胞毒性]

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引用本文的文献

[The role of interleukin-2 during the activation of cytotoxic T-lymphocytes].[白细胞介素-2在细胞毒性T淋巴细胞激活过程中的作用]

Klin Wochenschr. 1983 Jan 17;61(2):67-75. doi: 10.1007/BF01496657.

Mechanisms by which human gliomas may escape cellular immune attack.人类胶质瘤可能逃避细胞免疫攻击的机制。

Acta Neurochir (Wien). 1982;64(3-4):175-97. doi: 10.1007/BF01406052.

Enhancement of spontaneous killer cytotoxicity by soluble factor.可溶性因子增强自然杀伤细胞的细胞毒性

Clin Exp Immunol. 1980 Dec;42(3):589-96.

Biological effects of allogeneic effect factor on T lymphocytes: in vitro induction of cytotoxic T lymphocytes manifesting preferential lytic activity against H-2-identical tumor cells.同种异体效应因子对T淋巴细胞的生物学效应：体外诱导细胞毒性T淋巴细胞，其对H-2相同的肿瘤细胞表现出优先裂解活性。

Proc Natl Acad Sci U S A. 1979 Jul;76(7):3477-81. doi: 10.1073/pnas.76.7.3477.

In vitro sensitization of thymocytes. Role of H-21 I region determinants and cell-free mixed leukocyte culture supernates in generation of cytotoxic responses.胸腺细胞的体外致敏。H-21 I区决定簇和无细胞混合白细胞培养上清液在细胞毒性反应产生中的作用。

J Exp Med. 1978 Oct 1;148(4):953-62. doi: 10.1084/jem.148.4.953.

Cellular and genetic restrictions in the immunoregulatory activity of alpha-fetoprotein. II. Alpha-fetoprotein-induced suppression of cytotoxic T lymphocyte development.甲胎蛋白免疫调节活性中的细胞和遗传限制。II. 甲胎蛋白诱导的细胞毒性T淋巴细胞发育抑制

J Exp Med. 1978 Aug 1;148(2):360-72. doi: 10.1084/jem.148.2.360.

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