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基于超导双偶极模型的癌症病因与治疗统一理论

On a unified theory of cancer etiology and treatment based on the superconduction double-dipole model.

作者信息

Wolf A A

出版信息

Physiol Chem Phys. 1981;13(6):493-510.

PMID:7346865
Abstract

The human biological cell is a complex nonlinear system that behaves electrically as a double dipole. The nonlinear property of the cytoplasmic membrane permits is to divide; but it is the double-dipole property that motivates division and growth. Increasing the double-dipole moment increases speed of division. If the time required for division due to forces developed by the double-dipole becomes much less than the time needed for the chromatin material of the nucleus to properly develop and mature, defective genes will be formed, producing mutated daughter cells. Thus any stimulus that for prolonged periods increases the double-dipole moment can be responsible for producing mutated cells. One such stimulus is a "supercurrent" from an organic superconducting source. This supercurrent applied to tissue increases the cellular dipole moment, hence can produce an uncontrolled proliferation of biological cells giving rise to a tumor. In contrast, an injury current produces a controlled proliferation of embryonic cells in the traumatized area of the system. The latter proliferation is regulated by the negative feedback action of the host, which does not occur in the case of the supercurrent produced by an organic superconductor. Knowledge of the kind of organic superconductor involved, its transition temperature, and the critical magnetic field could make feasible a therapy aimed at terminating the offending supercurrent in the host.

摘要

人类生物细胞是一个复杂的非线性系统,其电行为表现为双偶极子。细胞质膜的非线性特性使其能够分裂;但促使分裂和生长的是双偶极子特性。增加双偶极矩会提高分裂速度。如果由双偶极子产生的力导致的分裂所需时间远小于细胞核染色质物质正常发育和成熟所需时间,就会形成有缺陷的基因,产生突变的子细胞。因此,任何长时间增加双偶极矩的刺激都可能导致产生突变细胞。一种这样的刺激是来自有机超导源的“超电流”。施加到组织上的这种超电流会增加细胞偶极矩,从而可能导致生物细胞不受控制地增殖,引发肿瘤。相比之下,损伤电流会使系统受创区域的胚胎细胞进行可控增殖。后者的增殖由宿主的负反馈作用调节,而有机超导体产生的超电流情况则不会发生这种调节。了解所涉及的有机超导体类型、其转变温度和临界磁场,可能使旨在终止宿主体内有害超电流的治疗成为可能。

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