Okuda K, Nakashima T, Okudaira M, Kage M, Aida Y, Omata M, Musha H, Futagawa S, Sugiura M, Kameda H
Liver. 1981 Dec;1(4):255-63. doi: 10.1111/j.1600-0676.1981.tb00041.x.
Hepatic venograms made in 40 authentic cases of idiopathic portal hypertension (Banti's syndrome) were compared with 13 normal venograms and venograms obtained in 88 cases of cirrhosis, and analyzed in the light of the pathological changes seen in 16 postmortem liver specimens. There were frequent anastomoses between hepatic vein radicles, approximation of middle-size branches to the liver surface, reduction in the angles between the main hepatic vein and its tributaries, and difficulty in opacifying portal vein branches in wedged retrograde portography. These angiographic alterations were corroborated by gross pathological findings which comprised displacement of middle-size hepatic vein branches closer to the liver surface and their approximation among themselves, and seem to be accounted for by the disappearance of liver parenchyma secondary to the peripheral portal circulatory failure.
对40例真性特发性门静脉高压症（班替氏综合征）患者的肝静脉造影片与13例正常肝静脉造影片以及88例肝硬化患者的肝静脉造影片进行了比较，并结合16例尸检肝脏标本的病理变化进行分析。肝静脉分支间常有吻合，中等大小分支靠近肝表面，肝主静脉与其分支间夹角减小，楔入式逆行门静脉造影时门静脉分支显影困难。这些血管造影改变得到大体病理结果的证实，包括中等大小肝静脉分支向肝表面移位且相互靠近，似乎是由于外周门静脉循环衰竭导致肝实质消失所致。
