肝脏线粒体腺嘌呤核苷酸池大小的体外改变：与呼吸功能的相关性 - Suppr

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In vitro alteration of the size of the liver mitochondrial adenine nucleotide pool: correlation with respiratory functions.

作者信息

Asimakis G K, Aprille J R

出版信息

Arch Biochem Biophys. 1980 Aug;203(1):307-16. doi: 10.1016/0003-9861(80)90181-2.

DOI:10.1016/0003-9861(80)90181-2

PMID:7406502

Abstract

摘要

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In vitro alteration of the size of the liver mitochondrial adenine nucleotide pool: correlation with respiratory functions.肝脏线粒体腺嘌呤核苷酸池大小的体外改变：与呼吸功能的相关性

Arch Biochem Biophys. 1980 Aug;203(1):307-16. doi: 10.1016/0003-9861(80)90181-2.

Adenine nucleotide pool size, adenine nucleotide translocase activity, and respiratory activity in newborn rabbit liver mitochondria.新生兔肝脏线粒体中的腺嘌呤核苷酸池大小、腺嘌呤核苷酸转位酶活性和呼吸活性。

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[Possible role of adenine nucleotide transport in regulating the respiration of rat liver mitochondria].腺嘌呤核苷酸转运在调节大鼠肝脏线粒体呼吸中的可能作用

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Adenine nucleotide transport during cardiac ischemia.心脏缺血期间的腺嘌呤核苷酸转运

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Determination of the free-energy difference of the adenine nucleotide translocator reaction in rat-liver mitochondria using intra- and extramitochondrial ATP-utilizing reactions.利用线粒体内外的ATP利用反应测定大鼠肝脏线粒体中腺嘌呤核苷酸转位酶反应的自由能差。

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Inhibition of Ca2(+)-induced large-amplitude swelling of liver and heart mitochondria by cyclosporin is probably caused by the inhibitor binding to mitochondrial-matrix peptidyl-prolyl cis-trans isomerase and preventing it interacting with the adenine nucleotide translocase.环孢素抑制Ca2+诱导的肝脏和心脏线粒体大幅度肿胀，可能是由于该抑制剂与线粒体基质肽基脯氨酰顺反异构酶结合，阻止其与腺嘌呤核苷酸转位酶相互作用所致。

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