红细胞生成性原卟啉病和迟发性皮肤卟啉病患者血清中趋化活性的产生。 - Suppr

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超能文献

Generation of chemotactic activity in serum from patients with erythropoietic protoporphyria and porphyria cutanea tarda.

作者信息

Lim H W, Perez H D, Poh-Fitzpatrick M, Goldstein I M, Gigli I

出版信息

N Engl J Med. 1981 Jan 22;304(4):212-6. doi: 10.1056/NEJM198101223040406.

DOI:10.1056/NEJM198101223040406

PMID:7442746

Abstract

摘要

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Generation of chemotactic activity in serum from patients with erythropoietic protoporphyria and porphyria cutanea tarda.红细胞生成性原卟啉病和迟发性皮肤卟啉病患者血清中趋化活性的产生。

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[Comparative morphological studies on porphyria cutanea tarda and erythropoietic protoporphyris].

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引用本文的文献

Recognized and Emerging Features of Erythropoietic and X-Linked Protoporphyria.红细胞生成性和X连锁原卟啉症的已知及新出现特征

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Complement-derived chemotactic activity is generated in human serum containing uroporphyrin after irradiation with 405 nm light.在含有尿卟啉的人血清经405nm光照射后，可产生补体衍生的趋化活性。

J Clin Invest. 1981 Apr;67(4):1072-7. doi: 10.1172/jci110119.

The role of complement in phototoxic reactions.补体在光毒性反应中的作用。

Springer Semin Immunopathol. 1981;4(3):209-19. doi: 10.1007/BF01892178.

Role of complement and polymorphonuclear cells in demethylchlortetracycline-induced phototoxicity in guinea pigs. Inhibition by decomplementation in vivo.补体和多形核细胞在豚鼠去甲金霉素诱导的光毒性中的作用。体内去补体作用的抑制。

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J Clin Invest. 1985 Aug;76(2):413-9. doi: 10.1172/JCI111987.

Activation of complement by 405-nm light in serum from porphyria cutanea tarda.迟发性皮肤卟啉症患者血清中405纳米光对补体的激活作用。

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