Nordenson I, Björksten B, Lundh B
Hum Genet. 1980;56(2):169-71. doi: 10.1007/BF00295689.
The frequency of aberrations in cultured lymphocytes from patients with Fanconi's anemia was significantly reduced when the cells were cocultivated with normal human lymphocytes. The results suggest that most of the chromosomal aberrations observed in cultured cells from Fanconi patients arise during cultivation and that the presence of normal cells prevents chromosomal damage by means of a hitherto unknown mechanism.
当范可尼贫血患者的培养淋巴细胞与正常人淋巴细胞共培养时，其染色体畸变频率显著降低。结果表明，在范可尼贫血患者培养细胞中观察到的大多数染色体畸变是在培养过程中产生的，并且正常细胞的存在通过一种迄今未知的机制防止了染色体损伤。
