Jampol L M, Condon P, Dizon-Moore R, Serjeant G, Schulman J A
Arch Ophthalmol. 1981 Feb;99(2):237-40. doi: 10.1001/archopht.1981.03930010239002.
In two patients with sickle cell disease (one hemoglobin SC and one hemoglobin SS), central retinal artery occlusions developed. In one case, the occlusion followed a retrobulbar injection of lidocaine hydrochloride. Although the central retinal artery reperfused in each patient, many secondary peripheral retinal arteriolar occlusions remained. During the subsequent days, multiple salmon-patch hemorrhages developed in the distribution of these occluded arterioles. In one patient, the salmon-patch hemorrhages evolved into atrophic schisis cavities. These unusual cases allowed us to document the origin of salmon-patch hemorrhages after peripheral retinal arteriolar occlusions. The development of the hemorrhages was a delayed phenomenon that occurred hours to days after the initial vascular occlusion. Reperfusion of the damaged ischemic vessels with a blowout of the wall of the vessels seems the most likely explanation for this phenomenon.
在两名镰状细胞病患者（一名血红蛋白SC型和一名血红蛋白SS型）中，发生了视网膜中央动脉阻塞。其中一例阻塞发生在球后注射盐酸利多卡因之后。尽管每名患者的视网膜中央动脉均再灌注，但仍残留许多继发性周边视网膜小动脉阻塞。在随后的几天里，这些阻塞小动脉分布区域出现了多处鲑鱼斑状出血。在一名患者中，鲑鱼斑状出血演变成了萎缩性视网膜劈裂腔。这些不同寻常的病例使我们能够记录周边视网膜小动脉阻塞后鲑鱼斑状出血的起源。出血的发生是一种延迟现象，在最初血管阻塞数小时至数天后出现。受损缺血血管的再灌注伴血管壁破裂似乎是对此现象最合理的解释。
