Broome J R
J R Nav Med Serv. 1995 Summer;81(2):120-6.
A minority of divers with neurological decompression illness (DCI) fail to improve with recompression treatment. This is particularly seen in cases where features of severe spinal cord injury develop soon after surfacing. Haemorrhage into the spinal cord is implicated in the pathogenesis of these cases, and evidence is presented that supports the view that the bleeding coincides with shrinkage of autochthonous bubbles. The role of hyperbaric oxygen therapy in the treatment of spinal cord DCI is discussed with reference to possible benefit in ischaemia-reperfusion (I-R) injury. Similarities and differences between the tissue injury of dysbaric and conventional spinal cord injury are outlined. The implications of advances in drug therapy for conventional spinal cord trauma are considered in the context of their potential application to treat neurological DCI.
少数患有神经减压病（DCI）的潜水员经再加压治疗后并无改善。这在浮出水面后不久即出现严重脊髓损伤特征的病例中尤为明显。脊髓出血与这些病例的发病机制有关，并且有证据支持出血与自身气泡收缩同时发生的观点。参考高压氧疗法对缺血再灌注（I-R）损伤可能的益处，讨论了其在脊髓DCI治疗中的作用。概述了减压性脊髓损伤与传统脊髓损伤在组织损伤方面的异同。在考虑药物治疗进展对传统脊髓创伤的潜在应用的背景下，探讨了其对治疗神经减压病的意义。
