• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

Cell damage in inflammatory and infectious sites might involve a coordinated "cross-talk" among oxidants, microbial haemolysins and ampiphiles, cationic proteins, phospholipases, fatty acids, proteinases and cytokines (an overview).

作者信息

Ginsburg I, Kohen R

机构信息

Department of Oral Biology, Hebrew University, Hadassah School of Dental Medicine, Jerusalem, Israel.

出版信息

Free Radic Res. 1995 Jun;22(6):489-517. doi: 10.3109/10715769509150323.

DOI:10.3109/10715769509150323
PMID:7633573
Abstract
摘要

相似文献

1
Cell damage in inflammatory and infectious sites might involve a coordinated "cross-talk" among oxidants, microbial haemolysins and ampiphiles, cationic proteins, phospholipases, fatty acids, proteinases and cytokines (an overview).炎症和感染部位的细胞损伤可能涉及氧化剂、微生物溶血素和两亲分子、阳离子蛋白、磷脂酶、脂肪酸、蛋白酶和细胞因子之间协调的“相互作用”（概述）。
Free Radic Res. 1995 Jun;22(6):489-517. doi: 10.3109/10715769509150323.
2
Could synergistic interactions among reactive oxygen species, proteinases, membrane-perforating enzymes, hydrolases, microbial hemolysins and cytokines be the main cause of tissue damage in infectious and inflammatory conditions?活性氧、蛋白酶、膜穿孔酶、水解酶、微生物溶血素和细胞因子之间的协同相互作用会是感染性和炎症性疾病中组织损伤的主要原因吗？
Med Hypotheses. 1998 Oct;51(4):337-46. doi: 10.1016/s0306-9877(98)90059-7.
3
Can we learn from the pathogenetic strategies of group A hemolytic streptococci how tissues are injured and organs fail in post-infectious and inflammatory sequelae?我们能否从A组溶血性链球菌的致病机制中了解到在感染后和炎症后遗症中组织是如何受损以及器官是如何衰竭的？
FEMS Immunol Med Microbiol. 1999 Sep;25(4):325-38. doi: 10.1111/j.1574-695X.1999.tb01357.x.
4
Multi-drug strategies are necessary to inhibit the synergistic mechanism causing tissue damage and organ failure in post infectious sequelae.多药策略对于抑制协同机制导致的感染后后遗症中的组织损伤和器官衰竭是必要的。
Inflammopharmacology. 1999;7(3):207-17. doi: 10.1007/s10787-999-0004-1.
5
Synergism among oxidants, proteinases, phospholipases, microbial hemolysins, cationic proteins, and cytokines.氧化剂、蛋白酶、磷脂酶、微生物溶血素、阳离子蛋白和细胞因子之间的协同作用。
Inflammation. 1992 Oct;16(5):519-38. doi: 10.1007/BF00918977.
6
Is there an optimal membrane protein content determined by membrane stability to lipid peroxidation? Disease and ageing consideration.是否存在由膜对脂质过氧化的稳定性所决定的最佳膜蛋白含量？对疾病和衰老的思考。
Biochem Soc Trans. 1996 Aug;24(3):376S. doi: 10.1042/bst024376s.
7
The role of bacteriolysis in the pathophysiology of inflammation, infection and post-infectious sequelae.细菌溶解在炎症、感染及感染后后遗症病理生理学中的作用。
APMIS. 2002 Nov;110(11):753-70. doi: 10.1034/j.1600-0463.2002.1101101.x.
8
Multiple roles of oxidants in the pathogenesis of asbestos-induced diseases.氧化剂在石棉所致疾病发病机制中的多重作用。
Free Radic Biol Med. 2003 May 1;34(9):1117-29. doi: 10.1016/s0891-5849(03)00060-1.
9
Male accessory gland infection: standardization of inflammatory parameters including cytokines.
Andrologia. 2003 Oct;35(5):300-3.
10
Oxidants and antioxidative defense.氧化剂与抗氧化防御
Hum Exp Toxicol. 2002 Feb;21(2):61-2. doi: 10.1191/0960327102ht210oa.

引用本文的文献

1
A Novel Hypothetical Approach to Explain the Mechanisms of Pathogenicity of Rheumatic Arthritis.一种解释风湿性关节炎发病机制的新型假说方法。
Mediterr J Rheumatol. 2021 May 28;32(2):112-117. doi: 10.31138/mjr.32.2.112. eCollection 2021 Jun.
2
Longitudinal patterns of cytokine expression at the individual level in humans after laparoscopic sleeve gastrectomy.腹腔镜袖状胃切除术后人个体水平细胞因子表达的纵向模式。
J Cell Mol Med. 2020 Jun;24(12):6622-6633. doi: 10.1111/jcmm.15309. Epub 2020 Apr 26.
3
Pro-inflammatory agents released by pathogens, dying host cells, and neutrophils act synergistically to destroy host tissues: a working hypothesis.
病原体、濒死宿主细胞和中性粒细胞释放的促炎因子协同作用以破坏宿主组织：一个工作假说。
J Inflamm Res. 2019 Jan 23;12:35-47. doi: 10.2147/JIR.S190007. eCollection 2019.
4
Uric acid disrupts hypochlorous acid production and the bactericidal activity of HL-60 cells.尿酸会破坏次氯酸的生成和 HL-60 细胞的杀菌活性。
Redox Biol. 2018 Jun;16:179-188. doi: 10.1016/j.redox.2018.02.020. Epub 2018 Mar 1.
5
Are histones real pathogenic agents in sepsis?组蛋白在脓毒症中是真正的病原体吗？
Nat Rev Immunol. 2018 Feb;18(2):148. doi: 10.1038/nri.2017.156. Epub 2017 Dec 27.
6
Serum histones as biomarkers of the severity of heatstroke in dogs.血清组蛋白可作为犬中暑严重程度的生物标志物。
Cell Stress Chaperones. 2017 Nov;22(6):903-910. doi: 10.1007/s12192-017-0817-6. Epub 2017 Jun 22.
7
The autodigestion hypothesis: Proteolytic receptor cleavage in rheological and cardiovascular cell dysfunction1.自消化假说：流变学和心血管细胞功能障碍中的蛋白水解性受体裂解1
Biorheology. 2016;53(5-6):179-191. doi: 10.3233/BIR-17131.
8
From amino acids polymers, antimicrobial peptides, and histones, to their possible role in the pathogenesis of septic shock: a historical perspective.从氨基酸聚合物、抗菌肽和组蛋白，到它们在脓毒性休克发病机制中的可能作用：历史视角。
J Inflamm Res. 2017 Feb 1;10:7-15. doi: 10.2147/JIR.S126150. eCollection 2017.
9
Circulating histones are major mediators of systemic inflammation and cellular injury in patients with acute liver failure.循环组蛋白是急性肝衰竭患者全身炎症和细胞损伤的主要介质。
Cell Death Dis. 2016 Sep 29;7(9):e2391. doi: 10.1038/cddis.2016.303.
10
Nuclear histones: major virulence factors or just additional early sepsis markers? A comment.核组蛋白：主要毒力因子还是仅仅是额外的早期脓毒症标志物？一则评论
Inflammopharmacology. 2016 Oct;24(5):287-289. doi: 10.1007/s10787-016-0279-y. Epub 2016 Sep 9.