Multiple sclerosis. More genes versus environment.
作者信息
Waksman B H
出版信息
Nature. 1995 Sep 14;377(6545):105-6. doi: 10.1038/377105a0.
Abstract
摘要
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本文引用的文献
1
Epidemiologic evidence for multiple sclerosis as an infection.多发性硬化症作为一种感染性疾病的流行病学证据。
Clin Microbiol Rev. 1993 Oct;6(4):382-427. doi: 10.1128/CMR.6.4.382.
2
Influence of infection on exacerbations of multiple sclerosis.感染对多发性硬化症病情加重的影响。
Ann Neurol. 1994;36 Suppl(Suppl):S25-8. doi: 10.1002/ana.410360709.
3
A genetic basis for familial aggregation in multiple sclerosis. Canadian Collaborative Study Group.多发性硬化症家族聚集现象的遗传基础。加拿大协作研究小组。
Nature. 1995 Sep 14;377(6545):150-1. doi: 10.1038/377150a0.
4
Molecular mimicry in T cell-mediated autoimmunity: viral peptides activate human T cell clones specific for myelin basic protein.T细胞介导的自身免疫中的分子模拟：病毒肽激活针对髓鞘碱性蛋白的人T细胞克隆。
Cell. 1995 Mar 10;80(5):695-705. doi: 10.1016/0092-8674(95)90348-8.
5
Adoptive transfer of EAE-like lesions from rats with coronavirus-induced demyelinating encephalomyelitis.从患有冠状病毒诱导的脱髓鞘性脑脊髓炎的大鼠身上进行实验性自身免疫性脑脊髓炎样病变的过继转移。
Nature. 1983;305(5930):150-3. doi: 10.1038/305150a0.
6
Measles encephalomyelitis--clinical and immunologic studies.麻疹脑脊髓炎——临床与免疫学研究
N Engl J Med. 1984 Jan 19;310(3):137-41. doi: 10.1056/NEJM198401193100301.
7
Exacerbations of multiple sclerosis in patients treated with gamma interferon.接受γ干扰素治疗的多发性硬化症患者病情加重情况。
Lancet. 1987 Apr 18;1(8538):893-5. doi: 10.1016/s0140-6736(87)92863-7.
8
Clinical viral infections and multiple sclerosis.临床病毒感染与多发性硬化症
Lancet. 1985 Jun 8;1(8441):1313-5. doi: 10.1016/s0140-6736(85)92801-6.
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Induction of autoimmune reactions to myelin basic protein in measles virus encephalitis in Lewis rats.在Lewis大鼠的麻疹病毒脑炎中诱导针对髓鞘碱性蛋白的自身免疫反应。
J Neuroimmunol. 1988 Jan;17(2):103-18. doi: 10.1016/0165-5728(88)90018-5.
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J Child Neurol. 1987 Oct;2(4):313-21. doi: 10.1177/088307388700200418.
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