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p53-based blood test for p53PIN3 and risk for sporadic ovarian cancer.

作者信息

Runnebaum I B, Tong X W, König R, Zhao H, Körner K, Atkinson E N, Kreienberg R, Kieback D G, Hong Z [corrected to Zhao H ]

出版信息

Lancet. 1995 Apr 15;345(8955):994. doi: 10.1016/s0140-6736(95)90745-9.

DOI:10.1016/s0140-6736(95)90745-9
PMID:7715326
Abstract
摘要

相似文献

1
p53-based blood test for p53PIN3 and risk for sporadic ovarian cancer.基于p53的血液检测用于检测p53PIN3及散发性卵巢癌风险。
Lancet. 1995 Apr 15;345(8955):994. doi: 10.1016/s0140-6736(95)90745-9.
2
p53 polymorphism in ovarian and bladder cancer.卵巢癌和膀胱癌中的p53基因多态性
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3
p53 polymorphism in ovarian and breast cancer.卵巢癌和乳腺癌中的p53基因多态性
Lancet. 1996 Feb 10;347(8998):393-4. doi: 10.1016/s0140-6736(96)90569-3.
4
TP53 intron 6 polymorphism and the risk of ovarian and breast cancer.TP53基因内含子6多态性与卵巢癌和乳腺癌风险
Br J Cancer. 1998 Feb;77(4):676-7. doi: 10.1038/bjc.1998.108.
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Loss of p53 and overexpression of EphA2 predict poor prognosis for ovarian cancer patients.p53缺失和EphA2过表达预示卵巢癌患者预后不良。
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6
No differences in p53 mutation frequencies between BRCA1-associated and sporadic ovarian cancers.BRCA1相关卵巢癌和散发性卵巢癌之间的p53突变频率没有差异。
Gynecol Oncol. 2004 Dec;95(3):430-6. doi: 10.1016/j.ygyno.2004.08.035.
7
Susceptibility to endometrial cancer: influence of allelism at p53, glutathione S-transferase (GSTM1 and GSTT1) and cytochrome P-450 (CYP1A1) loci.子宫内膜癌易感性：p53、谷胱甘肽S-转移酶（GSTM1和GSTT1）及细胞色素P-450（CYP1A1）基因座上等位性的影响
Br J Cancer. 1997;75(9):1385-8. doi: 10.1038/bjc.1997.235.
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[Inactivation of BRCA1, BRCA2 and p53 genes in sporadic ovarian cancer].
Ceska Gynekol. 2008 Oct;73(5):298-302.
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Distribution of p53 codon 72 polymorphisms in ovarian tumour patients and their prognostic significance in ovarian cancer patients.p53基因第72位密码子多态性在卵巢肿瘤患者中的分布及其对卵巢癌患者的预后意义。
Anticancer Res. 2002 May-Jun;22(3):1859-64.
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引用本文的文献

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Association of p53 (-16ins-pro) haplotype with the decreased risk of differentiated thyroid carcinoma in Iranian-Azeri patients.p53（-16ins-pro）单倍型与伊朗-阿塞拜疆患者分化型甲状腺癌风险降低的关联。
Pathol Oncol Res. 2015 Apr;21(2):449-54. doi: 10.1007/s12253-014-9846-y. Epub 2014 Nov 20.
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Association of TP53 PIN3 polymorphism with breast cancer in Moroccan population.摩洛哥人群中TP53 PIN3基因多态性与乳腺癌的关联。
Tumour Biol. 2014 Dec;35(12):12403-8. doi: 10.1007/s13277-014-2556-y. Epub 2014 Sep 9.
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Polymorphisms in p53 and the p53 pathway: roles in cancer susceptibility and response to treatment.
p53及p53信号通路中的多态性：在癌症易感性及治疗反应中的作用
J Cell Mol Med. 2009 Mar;13(3):440-53. doi: 10.1111/j.1582-4934.2008.00634.x.
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Construction of a high resolution linkage disequilibrium map to evaluate common genetic variation in TP53 and neural tube defect risk in an Irish population.构建高分辨率连锁不平衡图谱以评估爱尔兰人群中TP53基因的常见遗传变异与神经管缺陷风险的关系。
Am J Med Genet A. 2008 Oct 15;146A(20):2617-25. doi: 10.1002/ajmg.a.32504.
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Mutation and homozygous deletion analyses of genes that control the G1/S transition of the cell cycle in skin melanoma: p53, p21, p16 and p15.皮肤黑色素瘤中控制细胞周期G1/S转换的基因的突变和纯合缺失分析：p53、p21、p16和p15
Clin Transl Oncol. 2005 May;7(4):156-64. doi: 10.1007/BF02708753.
6
Intron variants of the p53 gene are associated with increased risk for ovarian cancer but not in carriers of BRCA1 or BRCA2 germline mutations.p53基因的内含子变异与卵巢癌风险增加相关，但在BRCA1或BRCA2种系突变携带者中并非如此。
Br J Cancer. 1999 Sep;81(1):179-83. doi: 10.1038/sj.bjc.6690669.
7
TP53 intron 6 polymorphism and the risk of ovarian and breast cancer.TP53基因内含子6多态性与卵巢癌和乳腺癌风险
Br J Cancer. 1998 Feb;77(4):676-7. doi: 10.1038/bjc.1998.108.
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Polymorphisms of H-ras-1 and p53 in breast cancer and lung cancer: a meta-analysis.乳腺癌和肺癌中H-ras-1和p53的多态性：一项荟萃分析。
Environ Health Perspect. 1997 Jun;105 Suppl 4(Suppl 4):919-26. doi: 10.1289/ehp.97105s4919.
9
Susceptibility to endometrial cancer: influence of allelism at p53, glutathione S-transferase (GSTM1 and GSTT1) and cytochrome P-450 (CYP1A1) loci.子宫内膜癌易感性：p53、谷胱甘肽S-转移酶（GSTM1和GSTT1）及细胞色素P-450（CYP1A1）基因座上等位性的影响
Br J Cancer. 1997;75(9):1385-8. doi: 10.1038/bjc.1997.235.
10
Molecular epidemiology in environmental health: the potential of tumor suppressor gene p53 as a biomarker.环境健康中的分子流行病学：肿瘤抑制基因p53作为生物标志物的潜力
Environ Health Perspect. 1997 Feb;105 Suppl 1(Suppl 1):155-63. doi: 10.1289/ehp.97105s1155.