Sato S, Tominaga T, Ohnishi T, Ohnishi S T
Philadelphia Biomedical Research Institute, King of Prussia, Pennsylvania 19406.
Ann N Y Acad Sci. 1994 Nov 17;738:369-73. doi: 10.1111/j.1749-6632.1994.tb21823.x.
In order to study the role of nitric oxide (NO) in rat brain ischemia, an in vivo spin trapping technique was applied. The results showed that NO was produced not only during ischemia but also during reperfusion. The role of NO production during ischemia may be to increase cerebral blood flow. The NO production during reperfusion seems to be related to tissue damage.
为了研究一氧化氮（NO）在大鼠脑缺血中的作用，应用了一种体内自旋捕获技术。结果表明，不仅在缺血期间而且在再灌注期间都会产生NO。缺血期间产生NO的作用可能是增加脑血流量。再灌注期间产生的NO似乎与组织损伤有关。
