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Determination of acetylator status in uraemia.

作者信息

Fine A, Sumner D J

出版信息

Br J Clin Pharmacol. 1975 Oct;2(5):475-6. doi: 10.1111/j.1365-2125.1975.tb00562.x.

DOI:10.1111/j.1365-2125.1975.tb00562.x
PMID:786360
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1402616/
Abstract
摘要

相似文献

1
Determination of acetylator status in uraemia.尿毒症患者乙酰化状态的测定。
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引用本文的文献

1
Phenelzine in urine: assay and relation to acetylator status.尿中苯乙肼的检测及其与乙酰化状态的关系。
Br J Clin Pharmacol. 1976 Aug;3(4):633-7. doi: 10.1111/j.1365-2125.1976.tb04887.x.
2
Determination of the acetylator phenotype and pharmacokinetics of some sulphonamides in man.人体中某些磺胺类药物乙酰化表型及药代动力学的测定
Clin Pharmacokinet. 1980 May-Jun;5(3):274-94. doi: 10.2165/00003088-198005030-00006.
3
Drug metabolites in renal failure: pharmacokinetic and clinical implications.肾衰竭中的药物代谢产物:药代动力学及临床意义
Clin Pharmacokinet. 1981 Sep-Oct;6(5):329-45. doi: 10.2165/00003088-198106050-00001.
4
Survey of the human acetylator polymorphism in spontaneous disorders.自发性疾病中人类乙酰化多态性的调查。
J Med Genet. 1984 Aug;21(4):243-53. doi: 10.1136/jmg.21.4.243.
5
Genetically determined variability in acetylation and oxidation. Therapeutic implications.乙酰化和氧化的基因决定变异性。治疗意义。
Drugs. 1985 Apr;29(4):342-75. doi: 10.2165/00003495-198529040-00003.
6
Drug acetylation in breast cancer.乳腺癌中的药物乙酰化作用。
Br J Cancer. 1989 Aug;60(2):236-7. doi: 10.1038/bjc.1989.260.
7
Polymorphic acetylation of sulphadimidine in normal and uraemic man.
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8
Clinical pharmacokinetics of isoniazid.异烟肼的临床药代动力学
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9
Disease and acetylation polymorphism.疾病与乙酰化多态性
Clin Pharmacokinet. 1977 May-Jun;2(3):182-97. doi: 10.2165/00003088-197702030-00003.

本文引用的文献

1
Sulphadimidine acetylation test for classification of patients as slow or rapid inactivators of isoniazid.用于将患者分类为异烟肼慢灭活者或快灭活者的磺胺二甲嘧啶乙酰化试验。
Br Med J. 1970 Aug 29;3(5721):495-7. doi: 10.1136/bmj.3.5721.495.
2
An improved and simplified method of detecting the acetylator phenotype.一种检测乙酰化酶表型的改良简化方法。
J Med Genet. 1969 Dec;6(4):405-7. doi: 10.1136/jmg.6.4.405.
3
Hepatotoxicity in rifampin-isoniazid treated patients related to their rate of isoniazid inactivation.
Chest. 1972 Jun;61(6):587-8. doi: 10.1378/chest.61.6_supplement.587.