Suemori A, Nakajima K, Kurane R, Nakamura Y
National Institute of Bioscience and Human-Technology (NIBH), Ibaraki, Japan.
FEMS Microbiol Lett. 1995 Jan 1;125(1):31-5. doi: 10.1111/j.1574-6968.1995.tb07331.x.
Rhodococcus erythropolis strain S1 uses the gentisate pathway to metabolize salicylate and m-hydroxybenzoate and the protocatechuate pathway to degrade p-hydroxybenzoate. m-Hydroxybenzoate 6-hydroxylase was induced by growth on m-hydroxybenzoate or gentisate, and salicylate 5-hydroxylase only by growth on salicylate. p-Hydroxybenzoate 3-hydroxylase could be induced only by growth on p-hydroxybenzoate. m-Hydroxybenzoate or p-hydroxybenzoate could repress the induction of salicylate 5-hydroxylase. Maleylpyruvate isomerase in the gentisate pathway did not require reduced glutathione.
红平红球菌菌株S1利用龙胆酸途径代谢水杨酸和间羟基苯甲酸，并利用原儿茶酸途径降解对羟基苯甲酸。间羟基苯甲酸6-羟化酶在间羟基苯甲酸或龙胆酸上生长时被诱导，而水杨酸5-羟化酶仅在水杨酸上生长时被诱导。对羟基苯甲酸3-羟化酶仅在对羟基苯甲酸上生长时可被诱导。间羟基苯甲酸或对羟基苯甲酸可抑制水杨酸5-羟化酶的诱导。龙胆酸途径中的马来酰丙酮酸异构酶不需要还原型谷胱甘肽。
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