Larsson D G, Hyllner S J, Haux C
Department of Zoophysiology, University of Göteborg, Sweden.
Gen Comp Endocrinol. 1994 Dec;96(3):445-50. doi: 10.1006/gcen.1994.1201.
Induction of vitelline envelope proteins by estradiol-17 beta was investigated in 14 teleost species from five systematic groups to assess whether estradiol-17 beta controls the synthesis of vitelline envelope proteins in teleost fish. Vitelline envelope proteins were detected in plasma from fish treated with estradiol-17 beta using three different antisera directed against vitelline envelope proteins from three teleosts. Induction of vitelline envelope proteins was demonstrated in 10 species. In 6 of these species males were available and used to demonstrate that the synthesis of vitelline envelope proteins is not restricted to the ovaries. The immunoreactivity of the proteins varied considerably within and between species. It is suggested that the synthesis of vitelline envelope proteins is controlled by estradiol-17 beta in the majority of teleost species.
为评估17β-雌二醇是否控制硬骨鱼卵黄膜蛋白的合成，研究人员在来自五个分类群的14种硬骨鱼中研究了17β-雌二醇对卵黄膜蛋白的诱导作用。使用针对三种硬骨鱼卵黄膜蛋白的三种不同抗血清，在经17β-雌二醇处理的鱼的血浆中检测到了卵黄膜蛋白。在10个物种中证实了卵黄膜蛋白的诱导作用。在其中6个物种中有雄性个体，并用于证明卵黄膜蛋白的合成并不局限于卵巢。这些蛋白质的免疫反应性在种内和种间有很大差异。提示在大多数硬骨鱼物种中，卵黄膜蛋白的合成受17β-雌二醇控制。
